Literature DB >> 26940227

Aspirin use and the risk of cholangiocarcinoma.

Jonggi Choi1, Hassan M Ghoz1, Thoetchai Peeraphatdit1,2, Esha Baichoo1,3, Benyam D Addissie1, William S Harmsen4, Terry M Therneau4, Janet E Olson5, Roongruedee Chaiteerakij1,6, Lewis R Roberts1.   

Abstract

UNLABELLED: Whether aspirin use is protective against cholangiocarcinoma (CCA) remains unclear. We determined the association between aspirin use and other risk factors for each CCA subtype individually. In a hospital-based case-control study, 2395 CCA cases (1169 intrahepatic, 995 perihilar, and 231 distal) seen at the Mayo Clinic, Rochester, MN, from 2000 through 2014 were enrolled. Controls selected from the Mayo Clinic Biobank were matched two to one with cases by age, sex, race, and residence (n = 4769). Associations between aspirin use, other risk factors, and CCA risk were determined. Aspirin was used by 591 (24.7%) CCA cases and 2129 (44.6%) controls. There was a significant inverse association of aspirin use with all CCA subtypes, with adjusted odds ratios (AORs) of 0.35 (95% confidence interval [CI], 0.29-0.42), 0.34 (95% CI 0.27-0.42), and 0.29 (95% CI 0.19-0.44) for intrahepatic, perihilar, and distal CCA, respectively (P < 0.001 for all). Primary sclerosing cholangitis was more strongly associated with perihilar (AOR = 453, 95% CI 104-999) than intrahepatic (AOR = 93.4, 95% CI 27.1-322) or distal (AOR = 34.0, 95% CI 3.6-323) CCA, whereas diabetes was more associated with distal (AOR = 4.2, 95% CI 2.5-7.0) than perihilar (AOR = 2.9, 95% CI 2.2-3.8) or intrahepatic (AOR = 2.5, 95% CI 2.0-3.2) CCA. Cirrhosis not related to primary sclerosing cholangitis was associated with both intrahepatic and perihilar CCA, with similar AORs of 14. Isolated inflammatory bowel disease without primary sclerosing cholangitis was not associated with any CCA subtype.
CONCLUSIONS: Aspirin use was significantly associated with a 2.7-fold to 3.6-fold decreased risk for the three CCA subtypes; our study demonstrates that individual risk factors confer risk of different CCA subtypes to different extents. (Hepatology 2016;64:785-796).
© 2016 by the American Association for the Study of Liver Diseases.

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Year:  2016        PMID: 26940227      PMCID: PMC5995727          DOI: 10.1002/hep.28529

Source DB:  PubMed          Journal:  Hepatology        ISSN: 0270-9139            Impact factor:   17.425


  32 in total

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4.  Aspirin use and risk of biliary tract cancer: a population-based study in Shanghai, China.

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5.  Differential expression of cyclooxygenase-2 (COX-2) in human bile duct epithelial cells and bile duct neoplasm.

Authors:  N Hayashi; H Yamamoto; N Hiraoka; K Dono; Y Ito; J Okami; M Kondo; H Nagano; K Umeshita; M Sakon; N Matsuura; S Nakamori; M Monden
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  33 in total

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Review 2.  Does Nonalcoholic Fatty Liver Disease Increase the Risk for Extrahepatic Malignancies?

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3.  Cholangiocarcinoma: from risk to prevention?

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4.  Association Between Aspirin and Cholangiocarcinoma in a Large Asian Cohort.

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6.  Risk Factors for Cholangiocarcinoma After Initial Hepatectomy for Intrahepatic Stones.

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7.  Body Mass Index, Diabetes and Intrahepatic Cholangiocarcinoma Risk: The Liver Cancer Pooling Project and Meta-analysis.

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8.  Epidemiology and Risk Factors of Cholangiocarcinoma.

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9.  Aspirin, Statins, Non-aspirin NSAIDs, Metformin, and the Risk of Biliary Cancer: A Swedish Population-Based Cohort Study.

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Review 10.  Mutational signatures and processes in hepatobiliary cancers.

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