| Literature DB >> 26930706 |
Jakob Engman1, Aurel Negrea1, Sara Sigurlásdóttir1, Miriam Geörg1, Jens Eriksson1, Olaspers Sara Eriksson1, Asaomi Kuwae1, Hong Sjölinder1, Ann-Beth Jonsson2.
Abstract
Neisseria meningitidis autoaggregation is an important step during attachment to human cells. Aggregation is mediated by type IV pili and can be modulated by accessory pilus proteins, such as PilX, and posttranslational modifications of the major pilus subunit PilE. The mechanisms underlying the regulation of aggregation remain poorly characterized. Polynucleotide phosphorylase (PNPase) is a 3'-5' exonuclease that is involved in RNA turnover and the regulation of small RNAs. In this study, we biochemically confirm that NMC0710 is the N. meningitidis PNPase, and we characterize its role in N. meningitidis pathogenesis. We show that deletion of the gene encoding PNPase leads to hyperaggregation and increased adhesion to epithelial cells. The aggregation induced was found to be dependent on pili and to be mediated by excessive pilus bundling. PNPase expression was induced following bacterial attachment to human cells. Deletion of PNPase led to global transcriptional changes and the differential regulation of 469 genes. We also demonstrate that PNPase is required for full virulence in an in vivo model of N. meningitidis infection. The present study shows that PNPase negatively affects aggregation, adhesion, and virulence in N. meningitidis.Entities:
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Year: 2016 PMID: 26930706 PMCID: PMC4862713 DOI: 10.1128/IAI.01463-15
Source DB: PubMed Journal: Infect Immun ISSN: 0019-9567 Impact factor: 3.441