Literature DB >> 26923819

Endothelial nitric oxide weakens arterial contractile responses and reduces blood pressure during early postnatal development in rats.

Svetlana I Sofronova1, Anna A Borzykh2, Dina K Gaynullina3, Ilya V Kuzmin4, Anastasia A Shvetsova4, Elena V Lukoshkova5, Olga S Tarasova4.   

Abstract

OBJECTIVE: During maturation the vascular system undergoes structural and functional remodeling. At the systemic level it results in a gradual increase of arterial blood pressure during postnatal ontogenesis. The mechanisms of maintaining the blood pressure at a comparatively low level during the early postnatal development are not completely understood. Recently we showed that the hindlimb arteries of young (1-2 wk-old) rats exhibited an enhanced endothelial NO-pathway activity, which weakened their contractile responsiveness compared to the arteries of adult rats. Here we tested the hypothesis that an increased tonic endothelial NO production can take place in the whole vascular system leading to a decreased level of systemic blood pressure in young rats. DESIGN AND METHODS: Segments of small mesenteric, saphenous, sural and intrarenal arteries were isolated from the young (2 wk-old), juvenile (4 wk-old) and adult (10-12 wk-old) male rats and tested in a wire isometric myograph. Anticontractile effect of NO was evaluated by the effects of NOS inhibitor L-NNA on both arterial spontaneous tone and constrictor responses to methoxamine (α1-adrenoceptor agonist). In addition, eNOS and arginase-2 mRNA expression in arterial preparations by qPCR and serum nitrite/nitrate levels by Griess reaction were estimated. Blood pressure with an intra-carotid artery catheter was measured in conscious rats.
RESULTS: In all arteries of 2 wk rats except the renal ones, L-NNA exposure resulted in a considerable tonic contraction and a remarkable enhancement of contractile responses to methoxamine. The effect of L-NNA gradually decreased with age and by 10-12 weeks became very small in the mesenteric arteries and disappeared in the sural and saphenous arteries. Although no difference in eNOS mRNA expression was found, the content of arginase-2 mRNA was significantly lower in young rats compared to adults. Serum levels of NO metabolites were two-fold higher in 2 wk-old rats than in adult rats. Along with that, arterial blood pressure was by half lower but rose more prominently after administration of l-NAME in young rats than in adults.
CONCLUSIONS: In young rats, tonic release of NO by the endothelium considerably weakens contractile responses of arteries supplying intestine, skin and skeletal muscles, which receive a high proportion of the cardiac output. Such anticontractile effect of NO can be an important mechanism responsible for the blood pressure reduction in immature circulatory system.
Copyright © 2016 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Arterial pressure; Endothelium; Nitric oxide; Postnatal development; Resistance arteries; eNOS

Mesh:

Substances:

Year:  2016        PMID: 26923819     DOI: 10.1016/j.niox.2016.02.005

Source DB:  PubMed          Journal:  Nitric Oxide        ISSN: 1089-8603            Impact factor:   4.427


  7 in total

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2.  Pathophysiological adaptations of resistance arteries in rat offspring exposed in utero to maternal obesity is associated with sex-specific epigenetic alterations.

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4.  Intrauterine L-NAME Exposure Weakens the Development of Sympathetic Innervation and Induces the Remodeling of Arterial Vessels in Two-Week-Old Rats.

Authors:  Ekaterina K Selivanova; Anastasia A Shvetsova; Anna A Borzykh; Dina K Gaynullina; Oxana O Kiryukhina; Elena V Lukoshkova; Viktoria M Potekhina; Vladislav S Kuzmin; Olga S Tarasova
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5.  MAPKs Are Highly Abundant but Do Not Contribute to α1-Adrenergic Contraction of Rat Saphenous Arteries in the Early Postnatal Period.

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Authors:  Anastasia A Shvetsova; Dina K Gaynullina; Nadine Schmidt; Peter Bugert; Elena V Lukoshkova; Olga S Tarasova; Rudolf Schubert
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  7 in total

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