Literature DB >> 26900377

Criminal Minds: Cav3.2 Channels Are the Culprits, but NMDAR Are the Co-Conspirators.

Huong Ha, John Huguenard.   

Abstract

Entities:  

Year:  2016        PMID： 26900377      PMCID： PMC4749115          DOI： 10.5698/1535-7597-16.1.36

Source DB:  PubMed          Journal:  Epilepsy Curr        ISSN： 1535-7511            Impact factor:   7.500


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  12 in total

1.  L-type voltage-gated calcium channels mediate NMDA-independent associative long-term potentiation at thalamic input synapses to the amygdala.

Authors:  M G Weisskopf; E P Bauer; J E LeDoux
Journal:  J Neurosci       Date:  1999-12-01       Impact factor: 6.167

2.  Lack of the burst firing of thalamocortical relay neurons and resistance to absence seizures in mice lacking alpha(1G) T-type Ca(2+) channels.

Authors:  D Kim; I Song; S Keum; T Lee; M J Jeong; S S Kim; M W McEnery; H S Shin
Journal:  Neuron       Date:  2001-07-19       Impact factor: 17.173

3.  A new drug for petit mal epilepsy.

Authors:  F T ZIMMERMAN; B B BURGEMEISTER
Journal:  Neurology       Date:  1958-10       Impact factor: 9.910

Review 4.  Bursts as a unit of neural information: making unreliable synapses reliable.

Authors:  J E Lisman
Journal:  Trends Neurosci       Date:  1997-01       Impact factor: 13.837

5.  The Ca(V)3.3 calcium channel is the major sleep spindle pacemaker in thalamus.

Authors:  Simone Astori; Ralf D Wimmer; Haydn M Prosser; Corrado Corti; Mauro Corsi; Nicolas Liaudet; Andrea Volterra; Paul Franken; John P Adelman; Anita Lüthi
Journal:  Proc Natl Acad Sci U S A       Date:  2011-08-01       Impact factor: 11.205

6.  Effects of Cav3.2 channel mutations linked to idiopathic generalized epilepsy.

Authors:  Houman Khosravani; Christopher Bladen; David B Parker; Terrance P Snutch; John E McRory; Gerald W Zamponi
Journal:  Ann Neurol       Date:  2005-05       Impact factor: 10.422

7.  Characterization of ethosuximide reduction of low-threshold calcium current in thalamic neurons.

Authors:  D A Coulter; J R Huguenard; D A Prince
Journal:  Ann Neurol       Date:  1989-06       Impact factor: 10.422

8.  Association between genetic variation of CACNA1H and childhood absence epilepsy.

Authors:  Yucai Chen; Jianjun Lu; Hong Pan; Yuehua Zhang; Husheng Wu; Keming Xu; Xiaoyan Liu; Yuwu Jiang; Xinhua Bao; Zhijian Yao; Keyue Ding; Wilson H Y Lo; Boqin Qiang; Piu Chan; Yan Shen; Xiru Wu
Journal:  Ann Neurol       Date:  2003-08       Impact factor: 10.422

9.  Mechanisms by which a CACNA1H mutation in epilepsy patients increases seizure susceptibility.

Authors:  Veit-Simon Eckle; Aleksandr Shcheglovitov; Iuliia Vitko; Deblina Dey; Chan Choo Yap; Bettina Winckler; Edward Perez-Reyes
Journal:  J Physiol       Date:  2013-11-25       Impact factor: 6.228

10.  CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy.

Authors:  Guangfu Wang; Genrieta Bochorishvili; Yucai Chen; Kathryn A Salvati; Peng Zhang; Steve J Dubel; Edward Perez-Reyes; Terrance P Snutch; Ruth L Stornetta; Karl Deisseroth; Alev Erisir; Slobodan M Todorovic; Jian-Hong Luo; Jaideep Kapur; Mark P Beenhakker; J Julius Zhu
Journal:  Genes Dev       Date:  2015-07-15       Impact factor: 12.890
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