Mamdouh Ahmed Gabr1, Mohamed Abd El-Raouf Tawfik2, Abd Allah Ahmed El-Sawy1. 1. Hepatogastroenterology and Endoscopy Unit, Internal Medicine Department, Tanta University Hospital, Tanta, Egypt. 2. Hepatogastroenterology and Endoscopy Unit, Internal Medicine Department, Tanta University Hospital, Tanta, Egypt. m_atawfik@hotmail.com.
Abstract
BACKGROUND AND STUDY AIMS: Acute upper gastrointestinal bleeding (AUGIB) in cirrhotic patients occurs mainly from esophageal and gastric varices; however, quite a large number of cirrhotic patients bleed from other sources as well. The aim of the present work is to determine the prevalence of non-variceal UGIB as well as its different causes among the cirrhotic portal hypertensive patients in Nile Delta. METHODS: Emergency upper gastrointestinal (UGI) endoscopy for AUGIB was done in 650 patients. Out of these patients, 550 (84.6%) patients who were proved to have cirrhosis were the subject of the present study. RESULTS: From all cirrhotic portal hypertensive patients, 415 (75.5%) bled from variceal sources (esophageal and gastric) while 135 (24.5%) of them bled from non-variceal sources. Among variceal sources of bleeding, esophageal varices were much more common than gastric varices. Peptic ulcer was the most common non-variceal source of bleeding. CONCLUSIONS: Non-variceal bleeding in cirrhosis was not frequent, and sources included peptic ulcer, portal hypertensive gastropathy, and erosive disease of the stomach and duodenum.
BACKGROUND AND STUDY AIMS: Acute upper gastrointestinal bleeding (AUGIB) in cirrhotic patients occurs mainly from esophageal and gastric varices; however, quite a large number of cirrhotic patients bleed from other sources as well. The aim of the present work is to determine the prevalence of non-variceal UGIB as well as its different causes among the cirrhotic portal hypertensivepatients in Nile Delta. METHODS:Emergency upper gastrointestinal (UGI) endoscopy for AUGIB was done in 650 patients. Out of these patients, 550 (84.6%) patients who were proved to have cirrhosis were the subject of the present study. RESULTS: From all cirrhotic portal hypertensivepatients, 415 (75.5%) bled from variceal sources (esophageal and gastric) while 135 (24.5%) of them bled from non-variceal sources. Among variceal sources of bleeding, esophageal varices were much more common than gastric varices. Peptic ulcer was the most common non-variceal source of bleeding. CONCLUSIONS: Non-variceal bleeding in cirrhosis was not frequent, and sources included peptic ulcer, portal hypertensive gastropathy, and erosive disease of the stomach and duodenum.
Authors: José Alberto González-González; Diego García-Compean; Genaro Vázquez-Elizondo; Aldo Garza-Galindo; Joel Omar Jáquez-Quintana; Héctor Maldonado-Garza Journal: Ann Hepatol Date: 2011 Jul-Sep Impact factor: 2.400
Authors: Pavel Svoboda; Michal Konecny; Arnost Martinek; Vladimir Hrabovsky; Vlastimil Prochazka; Jiri Ehrmann Journal: Biomed Pap Med Fac Univ Palacky Olomouc Czech Repub Date: 2012-05-25 Impact factor: 1.245