Literature DB >> 26879384

Prevalence and trend of neural tube defects in five counties in Shanxi province of Northern China, 2000 to 2014.

Jufen Liu1, Le Zhang1, Zhiwen Li1, Lei Jin1, Yali Zhang1, Rongwei Ye1, Jianmeng Liu1, Aiguo Ren1.   

Abstract

BACKGROUND: The prevalence of neural tube defects (NTDs) in northern China is among the highest in the world. A massive folic acid supplementation program as a specific countermeasure was introduced in 2009. Examining trends in NTD prevalence may provide evidence for future intervention.
METHODS: Data for 2000 to 2014 in five counties in northern China were obtained through a population-based birth defects surveillance system. All live births, stillbirths of over 20 gestational weeks, and pregnancy terminations because of NTDs at any gestational age were recorded. The prevalence of NTDs by gestational weeks (< 28 vs. ≥ 28), by calendar year, and by subtype was presented.
RESULTS: From 2000 to 2014, a total of 234,225 births and 2027 cases of NTDs were recorded. The prevalence of total NTDs was extremely high during 2000 to 2004, but it began to decrease continuously thereafter, from a peak of 120.0/10,000 in 2004 to a low of 31.5/10,000 in 2014. A significant decrease (60%) was observed from 78.8/10,000 in 2009 to 31.5/10,000 in 2014, 5 years after the folic acid supplementation program was introduced. All three major subtypes, namely anencephaly, spina bifida, and encephalocele, showed a decline over this period. Although the perinatal (≥ 28 gestational weeks) prevalence of NTDs decreased progressively, the pre-perinatal (< 28 gestational weeks) prevalence of NTDs remained high until 2011 and then decreased.
CONCLUSION: The prevalence of NTDs remains high despite a substantial and continuous decrease over the past 15 years. To further reduce NTD risk in the population, fortification staples with folic acid should be considered.
© 2016 Wiley Periodicals, Inc.

Entities:  

Keywords:  folic acid; neural tube defect; perinatal; prevalence; prevention

Mesh:

Substances:

Year:  2016        PMID: 26879384     DOI: 10.1002/bdra.23486

Source DB:  PubMed          Journal:  Birth Defects Res A Clin Mol Teratol        ISSN: 1542-0752


  31 in total

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2.  High levels of iron supplementation prevents neural tube defects in the Fpn1ffe mouse model.

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4.  Digenic variants of planar cell polarity genes in human neural tube defect patients.

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Review 5.  Genomic approaches to the assessment of human spina bifida risk.

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Journal:  Birth Defects Res       Date:  2017-01-30       Impact factor: 2.344

6.  Prevention of neural tube defects in Lrp2 mutant mouse embryos by folic acid supplementation.

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Journal:  Birth Defects Res       Date:  2017-01-20       Impact factor: 2.344

7.  Evaluating an intervention for neural tube defects in coal mining cites in China: a temporal and spatial analysis.

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8.  Formate rescues neural tube defects caused by mutations in Slc25a32.

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Journal:  Proc Natl Acad Sci U S A       Date:  2018-04-16       Impact factor: 11.205

9.  Folate-dependent methylation of septins governs ciliogenesis during neural tube closure.

Authors:  Manami Toriyama; Michinori Toriyama; John B Wallingford; Richard H Finnell
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10.  Prevalence and trend of isolated and complicated congenital hydrocephalus and preventive effect of folic acid in northern China, 2005-2015.

Authors:  Jufen Liu; Lei Jin; Zhiwen Li; Yali Zhang; Le Zhang; Linlin Wang; Aiguo Ren
Journal:  Metab Brain Dis       Date:  2018-02-01       Impact factor: 3.584

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