Literature DB >> 26837312

BCR-ABL1 expression, RT-qPCR and treatment decisions in chronic myeloid leukaemia.

Susan Latham1, Paul A Bartley1, Bradley Budgen1, David M Ross2, Elizabeth Hughes1, Susan Branford3, Deborah White4, Timothy P Hughes5, Alexander A Morley6.   

Abstract

AIMS: RT-qPCR is used to quantify minimal residual disease (MRD) in chronic myeloid leukaemia (CML) in order to make decisions on treatment, but its results depend on the level of BCR-ABL1 expression as well as leukaemic cell number. The aims of the study were to quantify inter-individual differences in expression level, to determine the relationship between expression level and response to treatment, and to investigate the effect of expression level on interpretation of the RT-qPCR result.
METHODS: BCR-ABL1 expression was studied in 248 samples from 65 patients with CML by determining the difference between MRD quantified by RT-qPCR and DNA-qPCR. The results were analysed statistically and by simple indicative modelling.
RESULTS: Inter-individual levels of expression approximated a normal distribution with an SD of 0.36 log. Expression at diagnosis correlated with expression during treatment. Response to treatment, as measured by the number of leukaemic cells after 3, 6 or 12 months of treatment, was not related to the level of expression. Indicative modelling suggested that interpretation of RT-qPCR results in relation to treatment guidelines could be affected by variation in expression when MRD was around 10% at 3 months and by both expression variation and Poisson variation when MRD was around or below the limit of detection of RT-qPCR.
CONCLUSIONS: Variation between individuals in expression of BCR-ABL1 can materially affect interpretation of the RT-qPCR when this test is used to make decisions on treatment. Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://www.bmj.com/company/products-services/rights-and-licensing/

Entities:  

Keywords:  CANCER GENETICS; CML; PCR

Mesh:

Substances:

Year:  2016        PMID: 26837312     DOI: 10.1136/jclinpath-2015-203538

Source DB:  PubMed          Journal:  J Clin Pathol        ISSN: 0021-9746            Impact factor:   3.411


  4 in total

1.  Reporting results for deep molecular responses in chronic myeloid leukemia.

Authors:  A A Morley
Journal:  Leukemia       Date:  2016-05-17       Impact factor: 11.528

2.  Digital PCR improves the quantitation of DMR and the selection of CML candidates to TKIs discontinuation.

Authors:  Simona Bernardi; Michele Malagola; Camilla Zanaglio; Nicola Polverelli; Elif Dereli Eke; Mariella D'Adda; Mirko Farina; Cristina Bucelli; Luigi Scaffidi; Eleonora Toffoletti; Clara Deambrogi; Fabio Stagno; Micaela Bergamaschi; Luca Franceschini; Elisabetta Abruzzese; Maria Domenica Divona; Marco Gobbi; Francesco Di Raimondo; Gianluca Gaidano; Mario Tiribelli; Massimiliano Bonifacio; Chiara Cattaneo; Alessandra Iurlo; Domenico Russo
Journal:  Cancer Med       Date:  2019-04-04       Impact factor: 4.452

3.  The validation of Short Interspersed Nuclear Elements (SINEs) as a RT-qPCR normalization strategy in a rodent model for temporal lobe epilepsy.

Authors:  René A J Crans; Jana Janssens; Sofie Daelemans; Elise Wouters; Robrecht Raedt; Debby Van Dam; Peter P De Deyn; Kathleen Van Craenenbroeck; Christophe P Stove
Journal:  PLoS One       Date:  2019-01-10       Impact factor: 3.240

4.  Hsa-miR-181a-5p, hsa-miR-182-5p, and hsa-miR-26a-5p as potential biomarkers for BCR-ABL1 among adult chronic myeloid leukemia treated with tyrosine kinase inhibitors at the molecular response.

Authors:  Aliza Mohd Yacob; Nor Asiah Muhamad; Kian Meng Chang; Hamidah Akmal Hisham; Yuslina Mat Yusoff; Latifah Ibrahim
Journal:  BMC Cancer       Date:  2022-03-26       Impact factor: 4.430

  4 in total

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