Literature DB >> 2682594

Function and metabolism of pancreatic beta-cells maintained in culture following experimentally induced damage.

D L Eizirik1, S Sandler.   

Abstract

Mesh:

Year:  1989        PMID: 2682594     DOI: 10.1111/j.1600-0773.1989.tb01149.x

Source DB:  PubMed          Journal:  Pharmacol Toxicol        ISSN: 0901-9928


× No keyword cloud information.
  6 in total

1.  Prolonged exposure of pancreatic islets isolated from "pre-diabetic" non-obese diabetic mice to a high glucose concentration does not impair beta-cell function.

Authors:  D L Eizirik; E Strandell; S Sandler
Journal:  Diabetologia       Date:  1991-01       Impact factor: 10.122

2.  Blockade of muscarinic transmission increases the frequency of diabetes after low-dose alloxan challenge in the mouse.

Authors:  B Ahrén; G Sundkvist; H Mulder; F Sundler
Journal:  Diabetologia       Date:  1996-04       Impact factor: 10.122

3.  Fine mapping of a region on chromosome 21q21.11-q22.3 showing linkage to type 1 diabetes.

Authors:  R Bergholdt; J Nerup; F Pociot
Journal:  J Med Genet       Date:  2005-01       Impact factor: 6.318

4.  Reversal of beta-cell suppression in vitro in pancreatic islets isolated from nonobese diabetic mice during the phase preceding insulin-dependent diabetes mellitus.

Authors:  E Strandell; D L Eizirik; S Sandler
Journal:  J Clin Invest       Date:  1990-06       Impact factor: 14.808

5.  Islet cell metabolism is reflected by the MTT (tetrazolium) colorimetric assay.

Authors:  D Janjic; C B Wollheim
Journal:  Diabetologia       Date:  1992-05       Impact factor: 10.122

6.  Long-term effects of alloxan in mice.

Authors:  B Ahrén; G Sundkvist
Journal:  Int J Pancreatol       Date:  1995-04
  6 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.