Literature DB >> 26824224

Determining carotid plaque vulnerability using ultrasound center frequency shifts.

Tobias Erlöv1, Magnus Cinthio2, Andreas Edsfeldt3, Simon Segstedt2, Nuno Dias4, Jan Nilsson3, Isabel Gonçalves5.   

Abstract

BACKGROUND: The leading cause of morbidity and mortality worldwide is atherosclerotic cardiovascular disease, most commonly caused by rupture of a high-risk plaque and subsequent thrombosis resulting in stroke, myocardial infarction or sudden death depending on the affected arterial territory. Accurate, non-invasive methods to identify such lesions known as vulnerable or high-risk plaques are currently sub-optimal. Our aim was to validate a new non-invasive ultrasound method to identify high-risk carotid plaques.
METHODS: We evaluated a new method based on the center frequency shift (CFS) of the ultrasound radio frequency data obtained from carotid plaques compared to a reference phantom. We evaluated the method both ex vivo, on 157 sections from 18 plaques, and in vivo, in 39 patients 1-day prior to carotid plaque removal, and correlated the data with histology.
RESULTS: The CFS correlated with a plaque vulnerability index based on histological areas stained for lipids, macrophages, hemorrhage, smooth muscle cells and collagen (r = -0.726, P = 1.7 × 10(-8)). Plaques with CFS below median had larger cores, more macrophages and were less rich in collagen in agreement with the definition of rupture-prone plaques. The accuracy to detect plaques with high vulnerability index was 78% (confidence interval (CI) 61-89%), with sensitivity 77% (CI 61-89%) and specificity 78% (CI 62-89%).
CONCLUSIONS: Our method is the first to characterize atherosclerotic plaque components that affect plaque vulnerability using CFS.
Copyright © 2016 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  Atherosclerosis; Imaging; Ultrasound; Vulnerable plaque

Mesh:

Substances:

Year:  2016        PMID: 26824224     DOI: 10.1016/j.atherosclerosis.2016.01.019

Source DB:  PubMed          Journal:  Atherosclerosis        ISSN: 0021-9150            Impact factor:   5.162


  5 in total

1.  Interferon regulatory factor-5-dependent CD11c+ macrophages contribute to the formation of rupture-prone atherosclerotic plaques.

Authors:  Andreas Edsfeldt; Maarten Swart; Pratibha Singh; Lea Dib; Jiangming Sun; Jennifer E Cole; Inhye Park; Dania Al-Sharify; Ana Persson; Mihaela Nitulescu; Patricia Das Neves Borges; Christina Kassiteridi; Michael E Goddard; Regent Lee; Petr Volkov; Marju Orho-Melander; Lars Maegdefessel; Jan Nilsson; Irina Udalova; Isabel Goncalves; Claudia Monaco
Journal:  Eur Heart J       Date:  2022-05-14       Impact factor: 35.855

2.  Serum Osteoprotegerin Is Associated With Calcified Carotid Plaque: A Strobe-Compliant Observational Study.

Authors:  Ami Kwon; Yun-Seok Choi; Yong-Won Choi; Woo-Baek Chung; Chul-Soo Park; Wook-Sung Chung; Man-Young Lee; Ho-Joong Youn
Journal:  Medicine (Baltimore)       Date:  2016-04       Impact factor: 1.889

3.  ADAMTS-7 is associated with a high-risk plaque phenotype in human atherosclerosis.

Authors:  Eva Bengtsson; Karin Hultman; Pontus Dunér; Giuseppe Asciutto; Peter Almgren; Marju Orho-Melander; Olle Melander; Jan Nilsson; Anna Hultgårdh-Nilsson; Isabel Gonçalves
Journal:  Sci Rep       Date:  2017-06-16       Impact factor: 4.379

4.  Altered metabolism distinguishes high-risk from stable carotid atherosclerotic plaques.

Authors:  Lukas Tomas; Andreas Edsfeldt; Inês G Mollet; Ljubica Perisic Matic; Cornelia Prehn; Jerzy Adamski; Gabrielle Paulsson-Berne; Ulf Hedin; Jan Nilsson; Eva Bengtsson; Isabel Gonçalves; Harry Björkbacka
Journal:  Eur Heart J       Date:  2018-06-21       Impact factor: 29.983

Review 5.  The conundrum of asymptomatic carotid stenosis-determinants of decision and evidence.

Authors:  José Fernandes E Fernandes; Luis Mendes Pedro; Isabel Gonçalves
Journal:  Ann Transl Med       Date:  2020-10
  5 in total

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