Literature DB >> 26820634

Altered interferon-γ response in patients with Q-fever fatigue syndrome.

Stephan P Keijmel1, Ruud P H Raijmakers2, Chantal P Bleeker-Rovers3, Jos W M van der Meer4, Mihai G Netea5, Teske Schoffelen6, Marcel van Deuren7.   

Abstract

OBJECTIVES: Whether immunological mechanisms underlie Q-fever fatigue syndrome (QFS) remains unclear. For acute Q-fever, the antigen-specific interferon-γ (IFNγ) response may be a useful tool for diagnosis, and the IFNγ/interleukin(IL)-2 production ratio may be a marker for chronic Q-fever and treatment monitoring. Here we explored the specific IFNγ production and IFNγ/IL-2 ratio in QFS patients.
METHODS: IFNγ and IL-2 production were tested in ex-vivo stimulated whole blood of QFS patients (n = 20), and compared to those previously determined in seropositive controls (n = 135), and chronic Q-fever patients (n = 28). Also, the correlation between patient characteristics and IFNγ, IL-2, and IFNγ/IL-2 ratio was determined.
RESULTS: QFS patients were younger (p < 0.001), but gender distribution was similar to seropositive controls and chronic Q-fever patients. Coxiella burnetii Nine Mile stimulation revealed a higher IFNγ production in QFS (median 319.5 pg/ml) than in seropositive controls (120 pg/ml, p < 0.01), but comparable to chronic Q-fever (2846 pg/ml). The IFNγ/IL-2 ratio was similar to that in seropositive controls, but lower than in chronic Q-fever patients (p < 0.01). Symptom duration was positively correlated with IL-2 production, and negatively correlated with the IFNγ/IL-2 ratio.
CONCLUSIONS: These results point to an altered cell-mediated immunity in QFS, and suggest a different immune response than in chronic Q-fever.
Copyright © 2016 The British Infection Association. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Cell-mediated immunity; Coxiella burnetii; Cytokines; Diagnosis; IFNγ; Interferon-gamma; Q-fever; Q-fever fatigue syndrome; QFS

Mesh:

Substances:

Year:  2016        PMID: 26820634     DOI: 10.1016/j.jinf.2016.01.004

Source DB:  PubMed          Journal:  J Infect        ISSN: 0163-4453            Impact factor:   6.072


  5 in total

Review 1.  Infection Elicited Autoimmunity and Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: An Explanatory Model.

Authors:  Jonas Blomberg; Carl-Gerhard Gottfries; Amal Elfaitouri; Muhammad Rizwan; Anders Rosén
Journal:  Front Immunol       Date:  2018-02-15       Impact factor: 7.561

2.  Interferon-γ and CXCL10 responses related to complaints in patients with Q fever fatigue syndrome.

Authors:  Ruud P H Raijmakers; Anne F M Jansen; Stephan P Keijmel; Teske Schoffelen; Anja Scholzen; Jos W M van der Meer; Leo A B Joosten; Mihai G Netea; Marcel van Deuren; Chantal P Bleeker-Rovers
Journal:  Eur J Clin Microbiol Infect Dis       Date:  2018-05-26       Impact factor: 3.267

3.  A possible role for mitochondrial-derived peptides humanin and MOTS-c in patients with Q fever fatigue syndrome and chronic fatigue syndrome.

Authors:  Ruud P H Raijmakers; Anne F M Jansen; Stephan P Keijmel; Rob Ter Horst; Megan E Roerink; Boris Novakovic; Leo A B Joosten; Jos W M van der Meer; Mihai G Netea; Chantal P Bleeker-Rovers
Journal:  J Transl Med       Date:  2019-05-14       Impact factor: 5.531

4.  A possible link between recurrent upper respiratory tract infections and lower cytokine production in patients with Q fever fatigue syndrome.

Authors:  Ruud P H Raijmakers; Anne F M Jansen; Stephan P Keijmel; Jos W M van der Meer; Leo A B Joosten; Mihai G Netea; Chantal P Bleeker-Rovers
Journal:  Eur J Immunol       Date:  2019-05-09       Impact factor: 5.532

5.  Multi-omics examination of Q fever fatigue syndrome identifies similarities with chronic fatigue syndrome.

Authors:  Ruud P H Raijmakers; Megan E Roerink; Anne F M Jansen; Stephan P Keijmel; Ranko Gacesa; Yang Li; Leo A B Joosten; Jos W M van der Meer; Mihai G Netea; Chantal P Bleeker-Rovers; Cheng-Jian Xu
Journal:  J Transl Med       Date:  2020-11-26       Impact factor: 5.531

  5 in total

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