| Literature DB >> 26810718 |
Ilya Ayzenberg1, Joanna Schöllhammer1, Robert Hoepner1, Kerstin Hellwig1, Marius Ringelstein2, Orhan Aktas2, Tania Kümpfel3, Markus Krumbholz3,4, Corinna Trebst5, Friedemann Paul6,7, Florence Pache6, Mark Obermann8, Lena Zeltner9, Matthias Schwab10, Achim Berthele11, Sven Jarius12, Ingo Kleiter13.
Abstract
Glatiramer acetate (GA) is an approved therapy for relapsing-remitting multiple sclerosis, but its efficacy for the prevention of attacks in neuromyelitis optica spectrum disorder (NMOSD) remains unknown. We did a multicenter retrospective analysis of GA-treated patients with NMOSD, identified through a national registry. Annualized relapse rate and expanded disability status scale (EDSS) were the main outcome measures. We identified 23 GA-treated patients (21 female, 16 aquaporin-4 antibody-positive). GA was given for <6 months in seven patients; reasons for stopping were relapses (n = 3), confirmation of NMOSD (n = 2) and side effects (n = 2). Of 16 patients treated ≥ 6 months with GA (15 female, 11 aquaporin-4 antibody-positive), 14 experienced at least one relapse. There was no reduction in the mean annualized relapse rate in the total group (1.9 ± 1.1 before vs. 1.8 ± 1.4 during GA therapy), as well as in those patients who were aquaporin-4 antibody-positive, or had a history of prior immunotherapy or not. The median EDSS increased (2.5 start vs. 3.5 finish of GA, P < 0.05). GA therapy was discontinued in 15/16 patients; reasons were therapeutic inefficacy in 13 and post-injection skin reactions in two patients. We conclude that GA is not beneficial for preventing attacks in most patients with NMOSD, particularly in aquaporin-4 antibody-positive cases.Entities:
Keywords: Aquaporin-4 antibody; Devic’s disease; Glatiramer acetate; Myelitis; Neuromyelitis optica spectrum disorder; Optic neuritis
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Year: 2016 PMID: 26810718 DOI: 10.1007/s00415-015-7991-1
Source DB: PubMed Journal: J Neurol ISSN: 0340-5354 Impact factor: 4.849