Literature DB >> 26784134

MIF reflects tissue damage rather than inflammation in post-cardiac arrest syndrome in a real life cohort.

Julia Pohl1, Christos Rammos1, Matthias Totzeck1, Pia Stock1, Malte Kelm2, Tienush Rassaf1, Peter Luedike3.   

Abstract

INTRODUCTION: Following successful resuscitation from cardiac arrest (CA), neurological impairment and other types of organ dysfunction cause significant morbidity and mortality-a condition termed post-cardiac arrest syndrome. Whole-body ischemia/reperfusion with oxygen debt activates immunologic and coagulation pathways increasing the risk of multiple organ failure and infection. We here examined the role of the pro-inflammatory cytokine macrophage migration inhibitory factor (MIF) in post-cardiac arrest syndrome.
METHODS: MIF plasma levels of n=16 patients with return of spontaneous circulation (ROSC) after CA were assessed with a previously validated method and compared to markers of systemic inflammation and cellular damage. ICU patients without former CA and healthy volunteers served as controls.
RESULTS: MIF levels in patients after ROSC were higher compared to those in healthy volunteers and ICU patients without CA. Kaplan-Meyer analysis revealed a distinctly elevated mortality since day one that further increased towards an elevated 60-days-mortality in patients with high plasma MIF. ROC curve identified plasma MIF as a predictor for mortality in patients after CA. Correlation with inflammatory parameters revealed that high MIF levels did not mirror post CA inflammatory syndrome, but distinctive cellular damage after ROSC as there were strong correlations with markers of cellular damage like LDH and GOT/GPT.
CONCLUSION: High MIF levels were associated with elevated 60-days-mortality and high MIF predicted mortality after CA. We found a close relation between circulating MIF levels and cellular damage, but not with an inflammatory syndrome.
Copyright © 2016 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  CPR; Cardiac arrest; ICU; MIF

Mesh:

Substances:

Year:  2016        PMID: 26784134     DOI: 10.1016/j.resuscitation.2015.12.015

Source DB:  PubMed          Journal:  Resuscitation        ISSN: 0300-9572            Impact factor:   5.262


  5 in total

1.  [MIF in post-resuscitation syndrome : No marker of inflammation, but of tissue damage].

Authors:  J Pohl
Journal:  Med Klin Intensivmed Notfmed       Date:  2016-08-03       Impact factor: 0.840

Review 2.  Assessing clinical implications and perspectives of the pathophysiological effects of erythrocytes and plasma free hemoglobin in autologous biologics for use in musculoskeletal regenerative medicine therapies. A review.

Authors:  Peter A Everts; Gerard A Malanga; Rowan V Paul; Joshua B Rothenberg; Natalie Stephens; Kenneth R Mautner
Journal:  Regen Ther       Date:  2019-05-10       Impact factor: 3.419

3.  Renal replacement therapy neutralizes elevated MIF levels in septic shock.

Authors:  Julia Pohl; Maria Papathanasiou; Martin Heisler; Pia Stock; Malte Kelm; Ulrike B Hendgen-Cotta; Tienush Rassaf; Peter Luedike
Journal:  J Intensive Care       Date:  2016-06-16

4.  Predictive potential of macrophage migration inhibitory factor (MIF) in patients with heart failure with preserved ejection fraction (HFpEF).

Authors:  Peter Luedike; Georgios Alatzides; Maria Papathanasiou; Martin Heisler; Julia Pohl; Nils Lehmann; Tienush Rassaf
Journal:  Eur J Med Res       Date:  2018-05-04       Impact factor: 2.175

5.  Myocardial Expression of Macrophage Migration Inhibitory Factor in Patients with Heart Failure.

Authors:  Julia Pohl; Ulrike B Hendgen-Cotta; Pia Stock; Peter Luedike; Hideo Andreas Baba; Markus Kamler; Tienush Rassaf
Journal:  J Clin Med       Date:  2017-10-13       Impact factor: 4.241

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.