Literature DB >> 26764016

Posterior reversible encephalopathy syndrome complicating diabetic ketoacidosis; an important treatable complication.

Rachel Jones1, Kasey Redler1, Jonathan Witherick1, Geraint Fuller1, Tripti Mahajan2, Benjamin R Wakerley1,3.   

Abstract

Development of acute neurological symptoms secondary to cerebral oedema is well described in diabetic ketoacidosis (DKA) and often has a poor prognosis. We present the clinical and radiological data of a 17-yr-old girl who developed cortical blindness, progressive encephalopathy, and seizures caused by posterior reversible encephalopathy syndrome (PRES) that developed after her DKA had resolved. Vasogenic oedema in PRES resolves if the underlying trigger is identified and eliminated. In this case, hypertension was identified as the likely precipitating factor and following treatment her vision and neurological symptoms rapidly improved. We suggest how recent DKA may have contributed to the development of PRES in this patient.
© 2016 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

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Keywords:  diabetic ketoacidosis; hypomagnesaemia; posterior reversible encephalopathy syndrome

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Year:  2016        PMID: 26764016     DOI: 10.1111/pedi.12362

Source DB:  PubMed          Journal:  Pediatr Diabetes        ISSN: 1399-543X            Impact factor:   4.866


  1 in total

1.  Can diabetic ketoacidosis (DKA) precipitate posterior reversible encephalopathy syndrome (PRES)?

Authors:  Ravi Sharma; Prateek Sharma; Varidh Katiyar; Zainab Vora; Hitesh Gurjar
Journal:  Childs Nerv Syst       Date:  2018-04-20       Impact factor: 1.475

  1 in total

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