Literature DB >> 26756591

Exploring the Validity of the Short Version of the Problem Behaviours Assessment (PBA-s) for Huntington's disease: A Rasch Analysis.

George McNally1, Hugh Rickards2, Mike Horton3, David Craufurd4.   

Abstract

BACKGROUND: The short version of the Problem Behaviours Assessment (PBA-s) is the recommended outcome measure for behavioural symptoms in Huntington's disease. Rasch analysis was used to further investigate the measurement limitations of the PBA-s.
OBJECTIVES: 1) To assess the psychometric properties of the 11 severity and frequency items within the PBA-s and 2) to determine the construct validity of using a total PBA-s score as a clinical outcome measure.
METHODS: PBA-s data for 517 participants from Enroll-HD were included in the Rasch analysis. Separate analyses were conducted for the severity and frequency items of the PBA-s, using RUMM2030 software. Achieving fit to the model provides supporting evidence that all items contribute to a single underlying latent trait. This property is defined as internal construct validity.
RESULTS: The total PBA-s severity score demonstrated several important limitations, including disordered response categories for all 11 severity items, local dependency and poor targeting. However, modifying the original five-point scoring system to a four-point system resulted in ordered response categories for seven of the severity items and achieved a good overall fit to the Rasch model. For the total PBA-s frequency score, fit to the model was not achieved even after amendments to the scoring system.
CONCLUSIONS: This study suggests that with reduction to a four-point scoring system, the total PBA-s severity score may be considered a valid clinical outcome measure. This study also suggests limitations in the use of a total PBA-s frequency score.

Entities:  

Keywords:  Enroll-HD; Huntington’s disease; Problem Behaviours Assessment; Rasch; psychometrics

Mesh:

Year:  2015        PMID: 26756591     DOI: 10.3233/JHD-150164

Source DB:  PubMed          Journal:  J Huntingtons Dis        ISSN: 1879-6397


  5 in total

1.  Specific patterns of brain alterations underlie distinct clinical profiles in Huntington's disease.

Authors:  Clara Garcia-Gorro; Alberto Llera; Saul Martinez-Horta; Jesus Perez-Perez; Jaime Kulisevsky; Nadia Rodriguez-Dechicha; Irene Vaquer; Susana Subira; Matilde Calopa; Esteban Muñoz; Pilar Santacruz; Jesus Ruiz-Idiago; Celia Mareca; Christian F Beckmann; Ruth de Diego-Balaguer; Estela Camara
Journal:  Neuroimage Clin       Date:  2019-06-15       Impact factor: 4.881

2.  Social cognition and quality of life in Huntington's disease.

Authors:  Clare M Eddy; Hugh Rickards
Journal:  Front Psychiatry       Date:  2022-08-24       Impact factor: 5.435

3.  Apathy and atrophy of subcortical brain structures in Huntington's disease: A two-year follow-up study.

Authors:  Verena Baake; Emma M Coppen; Erik van Duijn; Eve M Dumas; Simon J A van den Bogaard; Rachael I Scahill; Hans Johnson; Blair Leavitt; Alexandra Durr; Sarah J Tabrizi; David Craufurd; Raymund A C Roos
Journal:  Neuroimage Clin       Date:  2018-03-27       Impact factor: 4.881

4.  Microglia Activation in Basal Ganglia Is a Late Event in Huntington Disease Pathophysiology.

Authors:  Natalia P Rocha; Odelin Charron; Leigh B Latham; Gabriela D Colpo; Paolo Zanotti-Fregonara; Meixiang Yu; Leorah Freeman; Erin Furr Stimming; Antonio L Teixeira
Journal:  Neurol Neuroimmunol Neuroinflamm       Date:  2021-04-01

5.  Another Perspective on Huntington's Disease: Disease Burden in Family Members and Pre-Manifest HD When Compared to Genotype-Negative Participants from ENROLL-HD.

Authors:  Jannis Achenbach; Carsten Saft
Journal:  Brain Sci       Date:  2021-12-08
  5 in total

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