| Literature DB >> 26751395 |
Bertrand Isidor1,2, Sébastien Küry1, Jill A Rosenfeld3, Thomas Besnard1, Sébastien Schmitt1, Shelagh Joss4, Sally J Davies5, Robert Roger Lebel6, Alex Henderson7, Christian P Schaaf3, Haley E Streff3, Yaping Yang3, Vani Jain5, Nodoka Chida8, Xenia Latypova1, Cédric Le Caignec1,2, Benjamin Cogné1, Sandra Mercier1, Marie Vincent1, Estelle Colin9, Dominique Bonneau9, Anne-Sophie Denommé9, Philippe Parent10, Brigitte Gilbert-Dussardier11, Sylvie Odent12, Annick Toutain13, Amélie Piton14,15, Christian Dina16,17, Audrey Donnart16,17, Pierre Lindenbaum16,17, Eric Charpentier16,17, Richard Redon16,17, Kenji Iemura8, Masanori Ikeda8, Kozo Tanaka8, Stéphane Bézieau1.
Abstract
A rare syndromic form of intellectual disability with impaired speech was recently found associated with mutations in CHAMP1 (chromosome alignment-maintaining phosphoprotein 1), the protein product of which is directly involved in microtubule-kinetochore attachment. Through whole-exome sequencing in six unrelated nonconsanguineous families having a sporadic case of intellectual disability, we identified six novel de novo truncating mutations in CHAMP1: c.1880C>G p.(Ser627*), c.1489C>T; p.(Arg497*), c.1876_1877delAG; p.(Ser626Leufs*4), c.1043G>A; p.(Trp348*), c.1002G>A; p.(Trp334*), and c.958_959delCC; p.(Pro320*). Our clinical observations confirm the phenotypic homogeneity of the syndrome, which represents therefore a distinct clinical entity. Besides, our functional studies show that CHAMP1 protein variants are delocalized from chromatin and are unable to bind to two of its direct partners, POGZ and HP1. These data suggest a pathogenic mechanism of the CHAMP1-associated intellectual disability syndrome mediated by direct interacting partners of CHAMP1, several of which are involved in chromo/kinetochore-related disorders.Entities:
Keywords: POGZ; HP1; intellectual disability; CHAMP1; kinetochores; microcephaly; microtubules
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Year: 2016 PMID: 26751395 DOI: 10.1002/humu.22952
Source DB: PubMed Journal: Hum Mutat ISSN: 1059-7794 Impact factor: 4.878