Literature DB >> 26721601

Cigarette smoking behaviour and blood metabolomics.

Fangyi Gu1, Andriy Derkach2, Neal D Freedman2, Maria Teresa Landi2, Demetrius Albanes2, Stephanie J Weinstein2, Alison M Mondul3, Charles E Matthews2, Kristin A Guertin2, Qian Xiao2, Wei Zheng4, Xiao-Ou Shu4, Joshua N Sampson2, Steven C Moore2, Neil E Caporaso2.   

Abstract

BACKGROUND: Identifying circulating metabolites related to cigarette smoking may provide insight into the biological mechanisms of smoking-related diseases and the nature of addiction. However, previous studies are limited, generally small, and have largely targeted a priori metabolites.
METHODS: We examined associations between cigarette smoking and metabolites using an untargeted metabolomics approach in 892 men and women from four studies including participants from Italy, USA, China and Finland. We examined associations between individual log-transformed metabolites and two key smoking phenotypes (current smoking status and cigarettes per day [cig/day]) using linear regression. Fixed-effect meta-analysis was used to combine results across studies. Strict Bonferroni thresholds were used as our significance criteria. We further examined associated metabolites with other metrics of smoking behaviuor (current versus former, former versus never, smoking duration and years since quitting) in the US study.
RESULTS: We identified a total of 25 metabolites associated with smoking behaviours; 24 were associated with current smoking status and eight with cig/day. In addition to three well-established nicotine metabolites (cotinine, hydroxycotinine, cotinine N-oxide), we found an additional 12 xenobiotic metabolites involved in benzoatic (e.g. 3-ethylphenylsulphate) or xanthine metabolism (e.g. 1-methylurate), three amino acids (o-cresol sulphate, serotonin, indolepropionate), two lipids (scyllo-inositol, pregnenolone sulphate), four vitamins or cofactors [e.g. bilirubin (Z,Z)], and one carbohydrate (oxalate).
CONCLUSIONS: We identified associations between cigarette smoking and a diverse range of metabolites. Our findings, with further validation in future studies, have implications regarding aetiology and study design of smoking-related diseases. Published by Oxford University Press on behalf of the International Epidemiological Association 2015. This work is written by US Government employees and is in the public domain in the United States.

Entities:  

Keywords:  Smoking; bilirubin; metabolomics; non-targeted; o-cresol sulphate

Mesh:

Substances:

Year:  2015        PMID: 26721601      PMCID: PMC5100605          DOI: 10.1093/ije/dyv330

Source DB:  PubMed          Journal:  Int J Epidemiol        ISSN: 0300-5771            Impact factor:   7.196


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