| Literature DB >> 26720799 |
Seishi Kyoizumi1, Yoshiko Kubo1, Munechika Misumi2, Junko Kajimura1, Kengo Yoshida1, Tomonori Hayashi1, Kazue Imai1, Waka Ohishi3, Kei Nakachi1, Lauren F Young4, Jae-Hung Shieh5, Malcolm A Moore5, Marcel R M van den Brink4, Yoichiro Kusunoki1.
Abstract
It is not yet known whether hematopoietic stem and progenitor cells (HSPCs) are compromised in the aging population of atomic bomb (A-bomb) survivors after their exposure nearly 70 years ago. To address this, we evaluated age- and radiation-related changes in different subtypes of circulating HSPCs among the CD34-positive/lineage marker-negative (CD34(+)Lin(-)) cell population in 231 Hiroshima A-bomb survivors. We enumerated functional HSPC subtypes, including: cobblestone area-forming cells; long-term culture-initiating cells; erythroid burst-forming units; granulocyte and macrophage colony-forming units; and T-cell and natural killer cell progenitors using cell culture. We obtained the count of each HSPC subtype per unit volume of blood and the proportion of each HSPC subtype in CD34(+)Lin(-) cells to represent the lineage commitment trend. Multivariate analyses, using sex, age and radiation dose as variables, showed significantly decreased counts with age in the total CD34(+)Lin(-) cell population and all HSPC subtypes. As for the proportion, only T-cell progenitors decreased significantly with age, suggesting that the commitment to the T-cell lineage in HSPCs continuously declines with age throughout the lifetime. However, neither the CD34(+)Lin(-) cell population, nor HSPC subtypes showed significant radiation-induced dose-dependent changes in counts or proportions. Moreover, the correlations of the proportions among HSPC subtypes in the survivors properly revealed the hierarchy of lineage commitments. Taken together, our findings suggest that many years after exposure to radiation and with advancing age, the number and function of HSPCs in living survivors as a whole may have recovered to normal levels.Entities:
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Year: 2015 PMID: 26720799 PMCID: PMC5015444 DOI: 10.1667/RR14209.1
Source DB: PubMed Journal: Radiat Res ISSN: 0033-7587 Impact factor: 2.841