Literature DB >> 26700240

Effects of oxytocin on methamphetamine-seeking exacerbated by predator odor pre-exposure in rats.

Chantelle L Ferland1, Carmela M Reichel1, Jacqueline F McGinty2.   

Abstract

RATIONALE: The endogenous oxytocin system has emerged as an inhibitor of drug-seeking and stress in preclinical models.
OBJECTIVES: The goal of this study was to examine whether systemic oxytocin administration attenuated methamphetamine (METH)-seeking in rats pre-exposed to a predator odor threat.
METHODS: In Experiment 1, rats were exposed for 5 days to the predator odor, 2,5-dihydro-2,4,5-trimethylthiazoline (TMT), or saline before METH self-administration began. After extinction training, rats were injected with 1 mg/kg, ip oxytocin (OXT) or saline 30 min before a cue-induced reinstatement test followed by re-extinction and a TMT-induced reinstatement test. In Experiment 2, TMT pre-exposure was followed by 10 days of 1 mg/kg OXT or saline injections before METH self-administration, extinction, and a TMT-induced reinstatement test.
RESULTS: In Experiment 1, TMT pre-exposed rats that were injected with saline 30 min before reinstatement exhibited greater drug-seeking induced by conditioned cues or TMT than that exhibited by saline pre-exposed rats. A single injection of OXT 30 min before reinstatement suppressed METH-seeking in both saline- and TMT pre-exposed rats. In Experiment 2, TMT pre-exposed rats that received saline injections for 10 days prior to METH self-administration exhibited enhanced drug-seeking induced by TMT during stress-induced reinstatement. OXT injections for 10 days prior to METH self-administration blocked only the stress-induced exacerbation of drug-seeking in TMT pre-exposed rats.
CONCLUSIONS: These results support further research on the development of oxytocin as a novel therapeutic drug that has enduring effects on drug-seeking exacerbated by stress.

Entities:  

Keywords:  Addiction; Methamphetamine; Oxytocin; PTSD; Predator odor; Reinstatement; Self-administration; Stress; Substance use disorder

Mesh:

Substances:

Year:  2015        PMID: 26700240      PMCID: PMC5003622          DOI: 10.1007/s00213-015-4184-7

Source DB:  PubMed          Journal:  Psychopharmacology (Berl)        ISSN: 0033-3158            Impact factor:   4.530


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