Literature DB >> 26610798

KRAS Exon 2 Mutations as Prognostic Indicators in Advanced Colorectal Cancer in Clinical Practice: A Mono-Institutional Study.

Vincenzo Dadduzio1, Michele Basso2, Sabrina Rossi2, Tonia Cenci3, Sara Capodimonti3, Antonia Strippoli2, Armando Orlandi2, Eleonora Cerchiaro2, Giovanni Schinzari2, Alessandra Cassano2, Maurizio Martini3, Carlo Barone2.   

Abstract

BACKGROUND: Kirsten-Ras (KRAS) mutations are widely accepted negative predictive factors for anti-EGFR therapies in metastatic colorectal cancer (mCRC), while their prognostic significance is still under discussion.
OBJECTIVE: This mono-institutional retrospective study aims to investigate the real-life impact of exon 2 codon 12 and 13 mutations in mCRC.
METHODS: All mCRC patients treated at our institution between 2008 and 2014 carrying KRAS exon 2 mutations were included. The primary endpoint was to determine any significant difference in overall survival (OS) between codon 12 and 13 mutations. Secondary endpoints included progression-free survival (PFS), OS in both populations according to antiangiogenic treatment, and OS in liver-limited disease (LLD).
RESULTS: Of 620 mCRC patients, 218 carried KRAS exon 2 mutations (35.1%): 162 (26.1%) at codon 12 and 56 (9.0 %) at codon 13. Median OS results were similar: 32.0 months (codon 12) and 31.0 months (codon 13). PFS was also comparable, reaching 10.8 months in both populations. The addition of bevacizumab to chemotherapy conferred a trend toward survival advantage in codon 12 but not codon 13 mutation (p = 0.058). A high proportion of LLD patients underwent hepatic surgery with radical purpose (62.3%): in these patients, median OS has not yet been reached, while OS in non-LLD patients was 30.2 months.
CONCLUSIONS: No difference in OS between KRAS codon 12/13 mutated disease was found. This analysis showed a very prolonged OS for KRAS-mutated patients, even when LLD patients were excluded; OS of our real-life series favorably compares with OS of all-RAS wild-type patients in recent randomized studies.

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Year:  2016        PMID: 26610798     DOI: 10.1007/s40291-015-0178-8

Source DB:  PubMed          Journal:  Mol Diagn Ther        ISSN: 1177-1062            Impact factor:   4.074


  25 in total

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2.  Kirsten ras mutations in patients with colorectal cancer: the multicenter "RASCAL" study.

Authors:  H J Andreyev; A R Norman; D Cunningham; J R Oates; P A Clarke
Journal:  J Natl Cancer Inst       Date:  1998-05-06       Impact factor: 13.506

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Authors:  F Al-Mulla; E J Milner-White; J J Going; G D Birnie
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8.  Specific mutations in KRAS codons 12 and 13, and patient prognosis in 1075 BRAF wild-type colorectal cancers.

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9.  Fluorouracil, leucovorin, and irinotecan plus cetuximab treatment and RAS mutations in colorectal cancer.

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10.  BRAF mutation is a powerful prognostic factor in advanced and recurrent colorectal cancer.

Authors:  T Yokota; T Ura; N Shibata; D Takahari; K Shitara; M Nomura; C Kondo; A Mizota; S Utsunomiya; K Muro; Y Yatabe
Journal:  Br J Cancer       Date:  2011-02-01       Impact factor: 7.640

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Review 1.  Colorectal cancer in Saudi Arabia as the proof-of-principle model for implementing strategies of predictive, preventive, and personalized medicine in healthcare.

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2.  Deregulation of the miR-16-KRAS axis promotes colorectal cancer.

Authors:  Chaoying You; Hongwei Liang; Wu Sun; Jialu Li; Yanqing Liu; Qian Fan; Haiyang Zhang; Xin Yue; Jing Li; Xi Chen; Yi Ba
Journal:  Sci Rep       Date:  2016-11-18       Impact factor: 4.379

3.  KRAS exon 2 codon 13 mutation is associated with a better prognosis than codon 12 mutation following lung metastasectomy in colorectal cancer.

Authors:  Stéphane Renaud; Francesco Guerrera; Joseph Seitlinger; Lorena Costardi; Mickaël Schaeffer; Benoit Romain; Claudio Mossetti; Anne Claire-Voegeli; Pier Luigi Filosso; Michèle Legrain; Enrico Ruffini; Pierre-Emmanuel Falcoz; Alberto Oliaro; Gilbert Massard
Journal:  Oncotarget       Date:  2017-01-10
  3 in total

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