Literature DB >> 26589277

SomVarIUS: somatic variant identification from unpaired tissue samples.

Kyle S Smith1, Vinod K Yadav2, Shanshan Pei3, Daniel A Pollyea4, Craig T Jordan4, Subhajyoti De5.   

Abstract

MOTIVATION: Somatic variant calling typically requires paired tumor-normal tissue samples. Yet, paired normal tissues are not always available in clinical settings or for archival samples.
RESULTS: We present SomVarIUS, a computational method for detecting somatic variants using high throughput sequencing data from unpaired tissue samples. We evaluate the performance of the method using genomic data from synthetic and real tumor samples. SomVarIUS identifies somatic variants in exome-seq data of  ∼150 ×  coverage with at least 67.7% precision and 64.6% recall rates, when compared with paired-tissue somatic variant calls in real tumor samples. We demonstrate the utility of SomVarIUS by identifying somatic mutations in formalin-fixed samples, and tracking clonal dynamics of oncogenic mutations in targeted deep sequencing data from pre- and post-treatment leukemia samples.
AVAILABILITY AND IMPLEMENTATION: SomVarIUS is written in Python 2.7 and available at http://www.sjdlab.org/resources/ CONTACT: subhajyoti.de@ucdenver.edu SUPPLEMENTARY INFORMATION: Supplementary data are available at Bioinformatics online.
© The Author 2015. Published by Oxford University Press. All rights reserved. For Permissions, please e-mail: journals.permissions@oup.com.

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Year:  2015        PMID: 26589277     DOI: 10.1093/bioinformatics/btv685

Source DB:  PubMed          Journal:  Bioinformatics        ISSN: 1367-4803            Impact factor:   6.937


  18 in total

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Review 5.  A Survey of Computational Tools to Analyze and Interpret Whole Exome Sequencing Data.

Authors:  Jennifer D Hintzsche; William A Robinson; Aik Choon Tan
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6.  Clinically actionable mutation profiles in patients with cancer identified by whole-genome sequencing.

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10.  Identification of potentially oncogenic alterations from tumor-only samples reveals Fanconi anemia pathway mutations in bladder carcinomas.

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Journal:  NPJ Genom Med       Date:  2017-10-03       Impact factor: 8.617

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