Literature DB >> 26542466

TECPR2 mutations cause a new subtype of familial dysautonomia like hereditary sensory autonomic neuropathy with intellectual disability.

Gali Heimer1, Danit Oz-Levi2, Eran Eyal3, Shimon Edvardson4, Andreea Nissenkorn5, Elizabeth K Ruzzo6, Amir Szeinberg7, Channa Maayan8, Meir Mai-Zahav9, Ori Efrati10, Elon Pras11, Haike Reznik-Wolf12, Doron Lancet2, David B Goldstein13, Yair Anikster14, Stavit A Shalev15, Orly Elpeleg4, Bruria Ben Zeev16.   

Abstract

BACKGROUND: TECPR2 was first described as a disease causing gene when the c.3416delT frameshift mutation was found in five Jewish Bukharian patients with similar features. It was suggested to constitute a new subtype of complex hereditary spastic paraparesis (SPG49).
RESULTS: We report here 3 additional patients from unrelated non-Bukharian families, harboring two novel mutations (c.1319delT, c.C566T) in this gene. Accumulating clinical data clarifies that in addition to intellectual disability and evolving spasticity the main disabling feature of this unique disorder is autonomic-sensory neuropathy accompanied by chronic respiratory disease and paroxysmal autonomic events.
CONCLUSION: We suggest that the disease should therefore be classified as a new subtype of hereditary sensory-autonomic neuropathy. The discovery of additional mutations in non-Bukharian patients implies that this disease might be more common than previously appreciated and should therefore be considered in undiagnosed cases of intellectual disability with autonomic features and respiratory symptoms regardless of demographic origin.
Copyright © 2015 European Paediatric Neurology Society. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Autophagy; HSAN III; Hereditary spastic paraparesis; IKBKAP; Jewish Ashkenazi; TECPR2

Mesh:

Substances:

Year:  2015        PMID: 26542466     DOI: 10.1016/j.ejpn.2015.10.003

Source DB:  PubMed          Journal:  Eur J Paediatr Neurol        ISSN: 1090-3798            Impact factor:   3.140


  15 in total

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Review 2.  Autophagy in major human diseases.

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Review 3.  Genetic pain loss disorders.

Authors:  Annette Lischka; Petra Lassuthova; Arman Çakar; Christopher J Record; Jonas Van Lent; Jonathan Baets; Maike F Dohrn; Jan Senderek; Angelika Lampert; David L Bennett; John N Wood; Vincent Timmerman; Thorsten Hornemann; Michaela Auer-Grumbach; Yesim Parman; Christian A Hübner; Miriam Elbracht; Katja Eggermann; C Geoffrey Woods; James J Cox; Mary M Reilly; Ingo Kurth
Journal:  Nat Rev Dis Primers       Date:  2022-06-16       Impact factor: 65.038

4.  Expanding the Genotypic Spectrum of Congenital Sensory and Autonomic Neuropathies Using Whole-Exome Sequencing.

Authors:  Jose-Alberto Palma; Rachita Yadav; Dadi Gao; Lucy Norcliffe-Kaufmann; Susan Slaugenhaupt; Horacio Kaufmann
Journal:  Neurol Genet       Date:  2021-03-03

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Journal:  Curr Opin Neurobiol       Date:  2021-04-30       Impact factor: 7.070

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Authors:  Mansour Haidar; Vincent Timmerman
Journal:  Front Mol Neurosci       Date:  2017-05-11       Impact factor: 5.639

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Review 8.  TECPR2 mutation-associated respiratory dysregulation: more than central apnea.

Authors:  Pallavi P Patwari; Lisa F Wolfe; Girish D Sharma; Elizabeth Berry-Kravis
Journal:  J Clin Sleep Med       Date:  2020-06-15       Impact factor: 4.062

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Journal:  Autophagy       Date:  2020-11-29       Impact factor: 16.016

10.  A tecpr2 knockout mouse exhibits age-dependent neuroaxonal dystrophy associated with autophagosome accumulation.

Authors:  Bat-Chen Tamim-Yecheskel; Milana Fraiberg; Kamilya Kokabi; Saskia Freud; Oren Shatz; Letizia Marvaldi; Nemanja Subic; Ori Brenner; Michael Tsoory; Raya Eilam-Altstadter; Inbal Biton; Alon Savidor; Nili Dezorella; Gali Heimer; Christian Behrends; Bruria Ben-Zeev; Zvulun Elazar
Journal:  Autophagy       Date:  2020-12-10       Impact factor: 16.016

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