Literature DB >> 26523352

FcRγ-chain deficiency reduces the development of diet-induced obesity.

Lianne van Beek1,2, Irene O C M Vroegrijk2,3, Saeed Katiraei1,2, Mattijs M Heemskerk1,2, Andrea D van Dam2,3, Sander Kooijman2,3, Patrick C N Rensen2,3, Frits Koning4, J Sjef Verbeek1, Ko Willems van Dijk1,2,3, Vanessa van Harmelen1,2.   

Abstract

OBJECTIVE: Pathogenic immunoglobulins are produced during the development of obesity and contribute to the development of insulin resistance (IR). However, the mechanisms by which these antibodies affect IR are largely unknown. This study investigated whether Fc-receptors contribute to the development of diet-induced obesity and IR by studying FcRγ(-/-) mice that lack the γ-subunit necessary for signaling and cell surface expression of FcγR and FcεRI.
METHODS: FcRγ(-/-) and wild-type (WT) mice were fed a high-fat diet (HFD) to induce obesity. At 4 and 11 weeks, body weight and insulin sensitivity were measured, and adipose tissue (AT) inflammation was determined. Furthermore, intestinal triglyceride (TG) uptake and plasma TG clearance were determined, and gut microbiota composition was analyzed.
RESULTS: FcRγ(-/-) mice gained less weight after 11 weeks of HFD. They had reduced adiposity, adipose tissue inflammation, and IR. Interestingly, FcRγ(-/-) mice had higher lean mass compared to WT mice, which was associated with increased energy expenditure. Intestinal TG absorption was increased whereas plasma TG clearance was not affected in FcRγ(-/-) mice. Gut microbial composition differed significantly and might therefore have added to the observed phenotype.
CONCLUSIONS: FcRγ-chain deficiency reduces the development of diet-induced obesity, as well as associated AT inflammation and IR at 11 weeks of HFD.
© 2015 The Obesity Society.

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Year:  2015        PMID: 26523352     DOI: 10.1002/oby.21309

Source DB:  PubMed          Journal:  Obesity (Silver Spring)        ISSN: 1930-7381            Impact factor:   5.002


  8 in total

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2.  IgG is elevated in obese white adipose tissue but does not induce glucose intolerance via Fcγ-receptor or complement.

Authors:  A D van Dam; L van Beek; A C M Pronk; S M van den Berg; J Van den Bossche; M P J de Winther; F Koning; C van Kooten; P C N Rensen; M R Boon; J S Verbeek; K Willems van Dijk; V van Harmelen
Journal:  Int J Obes (Lond)       Date:  2017-08-30       Impact factor: 5.095

3.  A Restricted Role for FcγR in the Regulation of Adaptive Immunity.

Authors:  Marieke F Fransen; Hreinn Benonisson; Wendy W van Maren; Heng Sheng Sow; Cor Breukel; Margot M Linssen; Jill W C Claassens; Conny Brouwers; Jos van der Kaa; Marcel Camps; Jan Willem Kleinovink; Kelly K Vonk; Sandra van Heiningen; Ngaisah Klar; Lianne van Beek; Vanessa van Harmelen; Lucia Daxinger; Kutty S Nandakumar; Rikard Holmdahl; Chris Coward; Qingshun Lin; Sachiko Hirose; Daniela Salvatori; Thorbald van Hall; Cees van Kooten; Piero Mastroeni; Ferry Ossendorp; J Sjef Verbeek
Journal:  J Immunol       Date:  2018-03-09       Impact factor: 5.422

4.  BMT decreases HFD-induced weight gain associated with decreased preadipocyte number and insulin secretion.

Authors:  Saeed Katiraei; Lisa R Hoving; Lianne van Beek; Sharida Mohamedhoesein; Françoise Carlotti; Janna A van Diepen; Patrick C N Rensen; Mihai G Netea; Ko Willems van Dijk; Jimmy F P Berbée; Vanessa van Harmelen
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5.  Integrative analyses of biomarkers and pathways for adipose tissue after bariatric surgery.

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6.  LncRNA and mRNA expression profiles in brown adipose tissue of obesity-prone and obesity-resistant mice.

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Journal:  iScience       Date:  2022-08-06

8.  A novel immune-related genes signature after bariatric surgery is histologically associated with non-alcoholic fatty liver disease.

Authors:  Yancheng Song; Jan Zhang; Hexiang Wang; Dong Guo; Chentong Yuan; Bo Liu; Hao Zhong; Dongmei Li; Yu Li
Journal:  Adipocyte       Date:  2021-12       Impact factor: 4.534

  8 in total

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