Literature DB >> 26515423

JAK2 promotes brown adipose tissue function and is required for diet- and cold-induced thermogenesis in mice.

Sally Yu Shi1,2, Wei Zhang1,3, Cynthia T Luk1,2, Tharini Sivasubramaniyam1,2, Jara J Brunt1,2, Stephanie A Schroer1, Harsh R Desai1,4, Alexandra Majerski1, Minna Woo5,6,7.   

Abstract

AIMS/HYPOTHESIS: Non-shivering thermogenesis in adipose tissue can be activated by excessive energy intake or following cold exposure. The molecular mechanisms regulating this activation have not been fully elucidated. The Janus kinase (JAK) - signal transducer and activator of transcription (STAT) pathway mediates the signal transduction of numerous hormones and growth factors that regulate adipose tissue development and function, and may play a role in adaptive thermogenesis.
METHODS: We analysed mRNA and protein levels of uncoupling protein 1 (UCP1) and JAK2 in different adipose depots in response to metabolic and thermal stress. The in vivo role of JAK2 in adaptive thermogenesis was examined using mice with adipocyte-specific Jak2 deficiency (A-Jak2 KO).
RESULTS: We show in murine brown adipose tissue (BAT) that JAK2 is upregulated together with UCP1 in response to high-fat diet (HFD) feeding and cold exposure. In contrast to white adipose tissue, where JAK2 was dispensable for UCP1 induction, we identified an essential role for BAT JAK2 in diet- and cold-induced thermogenesis via mediating the thermogenic response to β-adrenergic stimulation. Accordingly, A-Jak2 KO mice were unable to upregulate BAT UCP1 following a HFD or after cold exposure. Therefore, A-Jak2 KO mice were cold intolerant and susceptible to HFD-induced obesity and diabetes. CONCLUSIONS/
INTERPRETATION: Taken together, our results suggest that JAK2 plays a critical role in BAT function and adaptive thermogenesis. Targeting the JAK-STAT pathway may be a novel therapeutic approach for the treatment of obesity and related metabolic disorders.

Entities:  

Keywords:  Adaptive thermogenesis; Brown adipose tissue; Diabetes; Glucose homeostasis; Janus kinase 2 (JAK2); Obesity; Uncoupling protein 1 (UCP1)

Mesh:

Substances:

Year:  2015        PMID: 26515423     DOI: 10.1007/s00125-015-3786-2

Source DB:  PubMed          Journal:  Diabetologia        ISSN: 0012-186X            Impact factor:   10.122


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7.  Identification of a rhodanine derivative BML-260 as a potent stimulator of UCP1 expression.

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9.  Cold exposure promotes obesity and impairs glucose homeostasis in mice subjected to a high‑fat diet.

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Journal:  Mol Med Rep       Date:  2018-08-10       Impact factor: 2.952

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Journal:  EBioMedicine       Date:  2018-10-20       Impact factor: 8.143

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