Literature DB >> 26482601

High prevalence of genetic alterations in early-onset epileptic encephalopathies associated with infantile movement disorders.

Yu Kobayashi1, Jun Tohyama2, Mitsuhiro Kato3, Noriyuki Akasaka1, Shinichi Magara1, Hideshi Kawashima1, Tsukasa Ohashi1, Hideaki Shiraishi4, Mitsuko Nakashima5, Hirotomo Saitsu5, Naomichi Matsumoto5.   

Abstract

OBJECTIVE: Recent studies have elucidated causative roles for genetic abnormalities in early-onset epileptic encephalopathies (EOEE). Accompanying characteristic features, in addition to seizures, have also been suggested to provide important clues for an early and accurate genetic diagnosis of affected patients. In this study, we investigated the underlying genetic causes in patients with EOEE associated with infantile movement disorders.
METHODS: We examined 11 patients with EOEE and involuntary movements (nine with West syndrome and two with nonsyndromic epileptic encephalopathy). All showed severe developmental delay, cognitive impairment, and involuntary movements such as chorea, ballism, dyskinesia or myoclonus, and hand stereotypies. We performed whole-exome sequencing of 10 patients, while the other patient underwent high-resolution melting analysis of candidate EOEE genes.
RESULTS: We identified mutations in CDKL5, SCN2A, SETD5, ALG13, and TBL1XR1 in seven patients with West syndrome, and in SCN1A and GRIN1 in the two patients with unclassified epileptic encephalopathy. All mutations were validated as de novo events. The genetic cause was undetermined in the remaining two patients.
CONCLUSIONS: We found pathogenic mutations in seven genes, in nine of 11 patients with EOEE and involuntary movements. Although the results of our study are preliminary because of the small number of patients, they nevertheless suggest that specific accompanying phenotypes such as hyperkinetic movements or hand stereotypies could be important in narrowing the disease spectrum and identifying causative genetic abnormalities.
Copyright © 2015 The Japanese Society of Child Neurology. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Epileptic encephalopathy; Hand stereotype; Hyperkinetic movement; Involuntary movement; West syndrome; Whole-exome sequencing

Mesh:

Substances:

Year:  2015        PMID: 26482601     DOI: 10.1016/j.braindev.2015.09.011

Source DB:  PubMed          Journal:  Brain Dev        ISSN: 0387-7604            Impact factor:   1.961


  24 in total

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2.  De Novo Variants in GRIA4 Lead to Intellectual Disability with or without Seizures and Gait Abnormalities.

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Review 6.  Perspectives on Glycosylation and Its Congenital Disorders.

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Authors:  Wienke H Galama; Sandra L J Verhaagen-van den Akker; Dirk J Lefeber; Ilse Feenstra; Aad Verrips
Journal:  JIMD Rep       Date:  2017-09-09

Review 8.  Progress in Understanding and Treating SCN2A-Mediated Disorders.

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10.  Clinical application of whole-exome sequencing: A retrospective, single-center study.

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