Literature DB >> 26449541

Interaction between variants in CLU and MS4A4E modulates Alzheimer's disease risk.

Mark T W Ebbert1, Kevin L Boehme1, Mark E Wadsworth1, Lyndsay A Staley1, Shubhabrata Mukherjee2, Paul K Crane2, Perry G Ridge1, John S K Kauwe3.   

Abstract

INTRODUCTION: Ebbert et al. reported gene-gene interactions between rs11136000-rs670139 (CLU-MS4A4E) and rs3865444-rs670139 (CD33-MS4A4E). We evaluate these interactions in the largest data set for an epistasis study.
METHODS: We tested interactions using 3837 cases and 4145 controls from Alzheimer's Disease Genetics Consortium using meta-analyses and permutation analyses. We repeated meta-analyses stratified by apolipoprotein E (APOE) ε4 status, estimated combined odds ratio (OR) and population attributable fraction (cPAF), and explored causal variants.
RESULTS: Results support the CLU-MS4A4E interaction and a dominant effect. An association between CLU-MS4A4E and APOE ε4 negative status exists. The estimated synergy factor, OR, and cPAF for rs11136000-rs670139 are 2.23, 2.45, and 8.0, respectively. We identified potential causal variants. DISCUSSION: We replicated the CLU-MS4A4E interaction in a large case-control series and observed APOE ε4 and possible dominant effect. The CLU-MS4A4E OR is higher than any Alzheimer's disease locus except APOE ε4, APP, and TREM2. We estimated an 8% decrease in Alzheimer's disease incidence without CLU-MS4A4E risk alleles and identified potential causal variants.
Copyright © 2016 The Alzheimer's Association. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  ADGC; ADNI; Alzheimer's disease; CD33; CLU; Epistasis; MS4A4E; Meta-analysis

Mesh:

Substances:

Year:  2015        PMID: 26449541      PMCID: PMC4744542          DOI: 10.1016/j.jalz.2015.08.163

Source DB:  PubMed          Journal:  Alzheimers Dement        ISSN: 1552-5260            Impact factor:   21.566


  41 in total

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  13 in total

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6.  Presenilin E318G variant and Alzheimer's disease risk: the Cache County study.

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Review 7.  Genetic Biomarkers on Age-Related Cognitive Decline.

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