| Literature DB >> 26445511 |
Andrea Viale1, Giulio F Draetta2.
Abstract
Tumors are metabolically heterogeneous, and subpopulations of tumorigenic cells have been recently described to rely more on mitochondrial respiration than glycolysis for energy production. In this issue, Sancho et al. (2015) demonstrate that MYC is a master switch regulating metabolic programs in different subpopulations of pancreatic tumor cells.Entities:
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Year: 2015 PMID： 26445511 DOI： 10.1016/j.cmet.2015.09.020
Source DB: PubMed Journal: Cell Metab ISSN： 1550-4131 Impact factor: 27.287