Literature DB >> 26440363

DNA double-strand breaks induced intractable glomerular fibrosis in renal allografts.

Yuki Matsui1, Yumi Sunatani2, Norifumi Hayashi1, Kazuaki Okino1, Yuki Okushi1, Kiyotaka Mukai1, Hiroki Adachi1, Hideki Yamaya1, Kuniyoshi Iwabuchi2, Hitoshi Yokoyama3.   

Abstract

BACKGROUNDS: The relationship between DNA damage and glomerular fibrosis in renal allografts remains unclear.
METHODS: We examined renal allograft specimens from 35 patients in which DNA double-strand breaks (DSBs) and glomerular fibrosis were detected by phospho-histone H2A.X (γ-H2AX) expression and collagen (COL) types III, IV, and VI accumulation. We also examined the in vitro relationship between DNA damage and COL accumulation by mitomycin C (MMc)-induced DNA damage in human glomerular endothelial cells (HRGEc).
RESULTS: The γ-H2AX and COL type VI, which mainly accumulated in the subendothelial and mesangial regions, were positively correlated with the duration of the post-renal transplant (RT) period. In multiple regression analysis, the duration of the post-RT period and cg in the Banff '07 classification were identified as a significant predictor of COL type VI accumulation and γ-H2AX expression in the glomerular capillaries. In addition, the γ-H2AX-positive area was also identified as a predictor of glomerular accumulation of COL type VI. COL type VI was detected in the cytoplasm of the HRGEc, which was secreted into the supernatant after MMc stimulation with γ-H2AX expression. The number of γ-H2AX (-)/COL type VI (+) cells was inversely associated with the number of γ-H2AX (+)/COL type VI (-) cells during 24-h MMc treatment.
CONCLUSIONS: Our findings suggest that the long-term RT induces DSBs and HRGEc-secreted COL type VI accumulation in the glomerular capillaries, which might progress to intractable glomerular fibrosis.

Entities:  

Keywords:  Collagen; DNA double-strand breaks; Endothelial dysfunction; Glomerular fibrosis; Renal allograft

Mesh:

Substances:

Year:  2015        PMID: 26440363     DOI: 10.1007/s10157-015-1174-3

Source DB:  PubMed          Journal:  Clin Exp Nephrol        ISSN: 1342-1751            Impact factor:   2.801


  29 in total

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