Literature DB >> 26430349

Factors for early death in rabies- the bitter truth.

Subramanian Senthilkumaran1, Narendra Nath Jena2, Sandeep B Gore3, Rishya Manikam4, Ponniah Thirumalaikolundusubramanian5.   

Abstract

Entities:  

Year:  2015        PMID: 26430349      PMCID: PMC4578207          DOI: 10.4103/0972-5229.164823

Source DB:  PubMed          Journal:  Indian J Crit Care Med        ISSN: 0972-5229


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Sir, Early death phenomenon in rabies reported by Sadeghi et al.[1] is informative and has long been recognized in patients whose incubation period is shorter than average in patients who were inadequately vaccinated with or without accompanying immunoglobulin and develop rabies.[2] However, we would like to recall other factors related to virus, patients, therapeutic agents, and care providers contributing to early death, so as to introduce appropriate measures to ensure quality in service and enforce patient safety. The high viral load inoculated at the site of bite, which infects motor endplates and motor axons without previous replication in the muscle and its virulence. Those related to patients are anatomical site of bite such as head and neck,[3] or bite in highly innervated areas such as fingers as noted in this case,[1] which has close proximity to central nervous system, and nonfeasibility to infiltrate adequate dose of human rabies immunoglobulin (HRIG). Next is the functional status of the immune system, which may be immune enhancement or “early death” phenomenon through B cells during the incubation period, and/or nonspecific immunosuppressive effect following rabies infection. Rabies nucleoprotein acts as a weak superantigen, and directly induces proliferation of human CD4 Th2 cells bearing the V8 TCR, which suppresses the immunity through polyclonal antibody response.[4] Matters related to therapeutic agents are questionable status of the potency of vaccine and HRIG, due to inherent technical constraints and electricity problems in countries such as India. Certain important factors related to health care providers are nonadherence to World Health Organization (WHO) postexposure prophylaxis protocol. Furthermore, health care providers should avoid prescribing antimalarials and other drugs that interfere with the effectiveness of rabies vaccine or HRIG. To overcome these, we need identification of other responsible factors so as to empower the health care providers for effective services and community health education.[5] Our motto shall be “no death due to rabies” through community education, early intervention, quality assurance of therapeutic agents, and adherence to WHO protocol.
  5 in total

1.  Rabies virus infects mouse and human lymphocytes and induces apoptosis.

Authors:  M I Thoulouze; M Lafage; J A Montano-Hirose; M Lafon
Journal:  J Virol       Date:  1997-10       Impact factor: 5.103

2.  Failures of post-exposure rabies prophylaxis.

Authors:  Henry Wilde
Journal:  Vaccine       Date:  2007-09-14       Impact factor: 3.641

3.  Acute rabies death mediated by antibody.

Authors:  B S Prabhakar; N Nathanson
Journal:  Nature       Date:  1981-04-16       Impact factor: 49.962

4.  Deaths from symptomatically identifiable furious rabies in India: a nationally representative mortality survey.

Authors:  Wilson Suraweera; Shaun K Morris; Rajesh Kumar; David A Warrell; Mary J Warrell; Prabhat Jha
Journal:  PLoS Negl Trop Dis       Date:  2012-10-04

5.  The rabies early death phenomenon: A report of ineffective administration of rabies vaccine during symptomatic disease.

Authors:  Mahmoud Sadeghi; Shams-Ali Hadizadeh Moallem; Elham Yousefi-Abdolmaleki; Mohammad Montazeri
Journal:  Indian J Crit Care Med       Date:  2015-07
  5 in total
  1 in total

1.  Rabies Treatment: Are We Anywhere Close to Cure?

Authors:  Subramanian Senthilkumaran; Namasivayam Balamurugan; Nanjundan Karthikeyan; Ponniah Thirumalaikolundusubramanian
Journal:  Indian J Crit Care Med       Date:  2018-03
  1 in total

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