Literature DB >> 26401930

Prolonged In Vivo Retention of a Cathepsin D Targeted Optical Contrast Agent in a Mouse Model of Alzheimer's Disease.

Jonatan A Snir1,2, Mojmir Suchy2,3, Keith St Lawrence1,4, Robert H E Hudson3, Stephen H Pasternak5,6, Robert Bartha1,2.   

Abstract

BACKGROUND: Cathepsin D (CatD) is a lysosomal protease that is elevated early in Alzheimer's disease (AD). We have previously developed a Targeted contrast agent (CA) to detect CatD activity in vivo, consisting of a magnetic resonance imaging/fluorescent moiety linked to a cell penetrating peptide (CPP) by means of a CatD cleavage site and have demonstrated its uptake in the brain of an AD mouse model.
OBJECTIVE: The purpose of this study was to characterize the in vivo retention of a near infra-red fluorescent dye labeled version of this CA.
METHODS: Six adult C57Bl/6 wild-type mice and six adult 5XFAD transgenic AD mice were studied using a small animal imaging system at five and twelve months of age using our novel Targeted CA, or two different control CAs; a Non-Targeted (lacking the CatD cleavage site) and a Non-Penetrating (lacking the CPP). Following intravenous CA administration, the optical signal was recorded within the brain and uptake and washout curves were measured and fitted to a one-phase exponential decay curve.
RESULTS: In all wild-type and 5XFAD mice, the washout of the Targeted CA that included a CPP domain was significantly slower than the washout of the Non-Penetrating and Non-Targeted CA. Furthermore, the washout of the CatD Targeted CA was significantly slower in the 5XFAD mice compared to the age matched wild-type controls (p <  0.05) at 5 and 12 months of age. Control CAs showed no differences in washout.
CONCLUSIONS: The prolonged retention of the CatD targeted CA in 5XFAD mice suggests this agent may be useful for AD detection.

Entities:  

Keywords:  Alzheimer’s disease; Cathepsin D; molecular imaging; optical imaging; transgenic mice

Mesh:

Substances:

Year:  2015        PMID: 26401930     DOI: 10.3233/JAD-150123

Source DB:  PubMed          Journal:  J Alzheimers Dis        ISSN: 1387-2877            Impact factor:   4.472


  4 in total

1.  Endogenous Apolipoprotein E (ApoE) Fragmentation Is Linked to Amyloid Pathology in Transgenic Mouse Models of Alzheimer's Disease.

Authors:  Anika Saul; Oliver Wirths
Journal:  Mol Neurobiol       Date:  2016-01-07       Impact factor: 5.590

2.  Inhibiting Extracellular Cathepsin D Reduces Hepatic Steatosis in Sprague⁻Dawley Rats .

Authors:  Princy Khurana; Tulasi Yadati; Sandeep Goyal; Atul Dolas; Tom Houben; Yvonne Oligschlaeger; Anil K Agarwal; Aditya Kulkarni; Ronit Shiri-Sverdlov
Journal:  Biomolecules       Date:  2019-05-04

3.  Detection of Active Caspase-3 in Mouse Models of Stroke and Alzheimer's Disease with a Novel Dual Positron Emission Tomography/Fluorescent Tracer [68Ga]Ga-TC3-OGDOTA.

Authors:  Valeriy G Ostapchenko; Jonatan Snir; Mojmir Suchy; Jue Fan; M Rebecca Cobb; Blaine A Chronik; Michael Kovacs; Vania F Prado; Robert H E Hudson; Stephen H Pasternak; Marco A M Prado; Robert Bartha
Journal:  Contrast Media Mol Imaging       Date:  2019-01-14       Impact factor: 3.161

4.  Increased Cathepsin D Correlates with Clinical Parameters in Newly Diagnosed Type 2 Diabetes.

Authors:  Lei Liu; Baoxian Chen; Xudong Zhang; Lun Tan; Dao Wen Wang
Journal:  Dis Markers       Date:  2017-12-10       Impact factor: 3.434

  4 in total

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