Warning: Undefined array key "mm" in /www/wwwroot/www.ai-bt.com/si.php on line 10 Deprecated: trim(): Passing null to parameter #1 ($string) of type string is deprecated in /www/wwwroot/www.ai-bt.com/si.php on line 10 Is iPLA2β a Novel Target for the Development of New Strategies to Alleviate Inflammatory Bowel Disease?

Literature DB >> 26391270

Is iPLA2β a Novel Target for the Development of New Strategies to Alleviate Inflammatory Bowel Disease?

Toni Petan¹, Igor Križaj^2,3.

Abstract

Entities: Gene

Mesh：

Substances：

Year: 2015 PMID： 26391270 DOI： 10.1007/s10620-015-3874-7

Source DB: PubMed Journal: Dig Dis Sci ISSN： 0163-2116 Impact factor: 3.199

× No keyword cloud information.

12 in total

Review 1. Recent progress in phospholipase A₂ research: from cells to animals to humans.

Authors: Makoto Murakami; Yoshitaka Taketomi; Yoshimi Miki; Hiroyasu Sato; Tetsuya Hirabayashi; Kei Yamamoto
Journal: Prog Lipid Res Date: 2010-12-24 Impact factor: 16.195

Review 2. Phospholipase A2 enzymes: physical structure, biological function, disease implication, chemical inhibition, and therapeutic intervention.

Authors: Edward A Dennis; Jian Cao; Yuan-Hao Hsu; Victoria Magrioti; George Kokotos
Journal: Chem Rev Date: 2011-09-12 Impact factor: 60.622

Review 3. The cell biology of the intestinal epithelium and its relation to inflammatory bowel disease.

Authors: J Jasper Deuring; Colin de Haar; Ernst J Kuipers; Maikel P Peppelenbosch; C Janneke van der Woude
Journal: Int J Biochem Cell Biol Date: 2013-01-03 Impact factor: 5.085

Review 4. Intestinal homeostasis and its breakdown in inflammatory bowel disease.

Authors: Kevin J Maloy; Fiona Powrie
Journal: Nature Date: 2011-06-15 Impact factor: 49.962

Review 5. Calcium-independent phospholipases A2 and their roles in biological processes and diseases.

Authors: Sasanka Ramanadham; Tomader Ali; Jason W Ashley; Robert N Bone; William D Hancock; Xiaoyong Lei
Journal: J Lipid Res Date: 2015-05-28 Impact factor: 5.922

6. Deficiency of Group VIA Phospholipase A2 (iPLA2β) Renders Susceptibility for Chemical-Induced Colitis.

Authors: Li Jiao; Johannes Inhoffen; Hongying Gan-Schreier; Sabine Tuma-Kellner; Wolfgang Stremmel; Zhiwei Sun; Walee Chamulitrat
Journal: Dig Dis Sci Date: 2015-07-17 Impact factor: 3.199

7. The role of PGE2 in intestinal inflammation and tumorigenesis.

Authors: David C Montrose; Masako Nakanishi; Robert C Murphy; Simona Zarini; Jeremy P McAleer; Anthony T Vella; Daniel W Rosenberg
Journal: Prostaglandins Other Lipid Mediat Date: 2014-10-22 Impact factor: 3.072

8. Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5'-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells.

Authors: Suzanne E Barbour; Phuong T Nguyen; Margaret Park; Bhargavi Emani; Xiaoyong Lei; Mamatha Kambalapalli; Jacqueline C Shultz; Dayanjan Wijesinghe; Charles E Chalfant; Sasanka Ramanadham
Journal: J Biol Chem Date: 2015-03-11 Impact factor: 5.157

Is iPLA2β a Novel Target for the Development of New Strategies to Alleviate Inflammatory Bowel Disease?

Review 1. Recent progress in phospholipase A₂ research: from cells to animals to humans.

Review 2. Phospholipase A2 enzymes: physical structure, biological function, disease implication, chemical inhibition, and therapeutic intervention.

Review 3. The cell biology of the intestinal epithelium and its relation to inflammatory bowel disease.

Review 4. Intestinal homeostasis and its breakdown in inflammatory bowel disease.

Review 5. Calcium-independent phospholipases A2 and their roles in biological processes and diseases.

6. Deficiency of Group VIA Phospholipase A2 (iPLA2β) Renders Susceptibility for Chemical-Induced Colitis.

7. The role of PGE2 in intestinal inflammation and tumorigenesis.

8. Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5'-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells.

Review 9. Group VIA Ca2+-independent phospholipase A2 (iPLA2beta) and its role in beta-cell programmed cell death.

Review 10. Biochemistry and physiology of mammalian secreted phospholipases A2.

Review 1. iPLA₂β and its role in male fertility, neurological disorders, metabolic disorders, and inflammation.

Is iPLA2β a Novel Target for the Development of New Strategies to Alleviate Inflammatory Bowel Disease?

Review 1. Recent progress in phospholipase A₂ research: from cells to animals to humans.

Review 2. Phospholipase A2 enzymes: physical structure, biological function, disease implication, chemical inhibition, and therapeutic intervention.

Review 3. The cell biology of the intestinal epithelium and its relation to inflammatory bowel disease.

Review 4. Intestinal homeostasis and its breakdown in inflammatory bowel disease.

Review 5. Calcium-independent phospholipases A2 and their roles in biological processes and diseases.

6. Deficiency of Group VIA Phospholipase A2 (iPLA2β) Renders Susceptibility for Chemical-Induced Colitis.

7. The role of PGE2 in intestinal inflammation and tumorigenesis.

8. Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5'-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells.

Review 9. Group VIA Ca2+-independent phospholipase A2 (iPLA2beta) and its role in beta-cell programmed cell death.

Review 10. Biochemistry and physiology of mammalian secreted phospholipases A2.

Review 1. iPLA2β and its role in male fertility, neurological disorders, metabolic disorders, and inflammation.

Review 1. iPLA₂β and its role in male fertility, neurological disorders, metabolic disorders, and inflammation.