Literature DB >> 26382057

Reduced expression of membrane-bound (m)RAGE is a biomarker of multiple sclerosis disease progression.

Zohara Sternberg1, Anne Chiotti1, Joseph Tario2, Trevor Chichelli1, Neel Patel3, Kailash Chadha3, Jinhee Yu4, Yuval Karmon1.   

Abstract

OBJECTIVES: This study is one in series measuring RAGE axis (receptor for advanced glycation end products, its isoforms, and ligands) as a biomarker in multiple sclerosis (MS). We identified and quantified membrane-bound RAGE (mRAGE) expression levels on freshly isolated PBMCs and its subpopulation (monocytes and T cells), and determined the relationship between mRAGE expression levels and MS disease severity.
MATERIALS AND METHODS: mRAGE expression was determined for 28 MS patients and 16HCs, by flow cytometry, using fluorochrome unconjugated primary RAGE monoclonal antibody and a polyclonal secondary antibody conjugated to R-Phycoerythrin (PE).
RESULTS: After adjusting for multiple comparisons and correcting for group differences in age and gender, MS patients showed higher percentages of mRAGE-positive on PBMCs (12.4±2.1 vs. 4.08±0.8, P=0.02), monocytes (37.4±5.8 vs. 20.1±5.0, P=0.08) and T cells (4.1±1.2 vs. 2.1±0.3, P=0.05). SPMS patients' showed lower percentages of RAGE-positive monocytes (13.7±5.5 vs. 49.5±6.6, P=0.0006) and RAGE-positive T cells (4.1±1.8 vs. 6.6±1.5, P=0.04) than RRMS patients. We observed a negative relationship between the percentages of mRAGE-positive PBMCs and MS severity scale (MSSS) (r=-0.39, P=0.04), monocytes and EDSS (r=-0.48, P=0.01), monocytes and MSSS (r=-0.58, P=0.001), and T cells and MSSS (r=-0.40, P=0.04). Monocytes expression of mRAGE showed 0.811 area under the curve (95% CI: 0.64-0.98) sensitivity/specificity for MSSS.
CONCLUSION: The reduced mRAGE expression on PBMCs in general, and on monocytes in particular, can be used as biomarker of MS disease severity and progression.
Copyright © 2015 Elsevier GmbH. All rights reserved.

Entities:  

Keywords:  Advanced glycation; Biomarker; Cell surface expression; Disease severity; Flow cytometry; Mononuclear cells

Mesh:

Substances:

Year:  2015        PMID: 26382057     DOI: 10.1016/j.imbio.2015.09.007

Source DB:  PubMed          Journal:  Immunobiology        ISSN: 0171-2985            Impact factor:   3.144


  5 in total

Review 1.  Role of RAGE in the Pathogenesis of Neurological Disorders.

Authors:  Judyta Juranek; Konark Mukherjee; Bernard Kordas; Michał Załęcki; Agnieszka Korytko; Kamila Zglejc-Waszak; Jarosław Szuszkiewicz; Marta Banach
Journal:  Neurosci Bull       Date:  2022-06-21       Impact factor: 5.271

Review 2.  GANAB and N-Glycans Substrates Are Relevant in Human Physiology, Polycystic Pathology and Multiple Sclerosis: A Review.

Authors:  Roberto De Masi; Stefania Orlando
Journal:  Int J Mol Sci       Date:  2022-07-01       Impact factor: 6.208

3.  Inflammatory state exists in familial amyloid polyneuropathy that may be triggered by mutated transthyretin.

Authors:  Genki Suenaga; Tokunori Ikeda; Teruaki Masuda; Hiroaki Motokawa; Taro Yamashita; Kotaro Takamatsu; Yohei Misumi; Mitsuharu Ueda; Hirotaka Matsui; Satoru Senju; Yukio Ando
Journal:  Sci Rep       Date:  2017-05-08       Impact factor: 4.379

Review 4.  Methylglyoxal-Derived Advanced Glycation Endproducts in Multiple Sclerosis.

Authors:  Suzan Wetzels; Kristiaan Wouters; Casper G Schalkwijk; Tim Vanmierlo; Jerome J A Hendriks
Journal:  Int J Mol Sci       Date:  2017-02-15       Impact factor: 5.923

Review 5.  Pathophysiology of RAGE in inflammatory diseases.

Authors:  Hanbing Dong; Yue Zhang; Yu Huang; Hui Deng
Journal:  Front Immunol       Date:  2022-07-29       Impact factor: 8.786

  5 in total

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