Literature DB >> 26375922

Effects of MTHFR c.677C>T, F2 c.20210G>A and F5 Leiden Polymorphisms in Gastroschisis.

Akhmad Makhmudi1, Ahmad Hamim Sadewa2, Teguh Aryandono3, Sumantra Chatterjee4, Hugo A Heij5.   

Abstract

BACKGROUND: Gastroschisis is a developmental disorder involving the extrusion of fetal intestines through a defect in the abdominal wall. The mechanism is presumed to be a dual vascular/thrombotic pathogenesis, where normal right umbilical vein involution forms a possible site for thrombosis adjacent to the umbilical ring.
PURPOSE: The aim of this study was to demonstrate that the 3 common prothrombotic polymorphisms, MTHFR c.677C>T, F2 c.20210G>A, and F5 Leiden, were elevated in frequency in Indonesian gastroschisis patients.
MATERIAL AND METHODS: Three genetic markers were investigated in 46 patients with gastroschisis and 89 ethnicity-matched controls for association studies using polymerase chain reaction-restriction fragment length polymorphisms (PCR-RFLP) or TaqMan Genotyping Assays on genomic DNA.
RESULTS: MTHFR c.677C>T showed a significant association with gastroschisis (OR = 2.1, 95% CI = 1.13-3.86; p = .018) but no affected infants had risk alleles for either F2 c.20210G>A or F5 Leiden. Further, the frequency of MTHFR risk allele (T) in patients with maternal age <25 years is marginally significant higher than those in cases with maternal age ≥25 years (p = .069) with an OR of 2.7 (95% CI = 0.90-8.07).
CONCLUSIONS: MTHFR is a common susceptibility factor for gastroschisis in Indonesia. The increased gastroschisis risk in offspring of younger maternal age suggests the thrombotic pathogenesis model. A founder effect is the most likely explanation for the rarity of the F2 and F5 Leiden polymorphisms in Indonesian population.

Entities:  

Keywords:  Gastroschisis; Indonesia; founder effect; maternal age; prothrombotic polymorphisms; vascular/thrombotic pathogenesis

Mesh:

Substances:

Year:  2015        PMID: 26375922     DOI: 10.3109/08941939.2015.1077908

Source DB:  PubMed          Journal:  J Invest Surg        ISSN: 0894-1939            Impact factor:   2.533


  4 in total

1.  Effects of SEMA3 polymorphisms in Hirschsprung disease patients.

Authors:  Akhmad Makhmudi; Nunik Agustriani
Journal:  Pediatr Surg Int       Date:  2016-07-28       Impact factor: 1.827

Review 2.  Genetic variants conferring susceptibility to gastroschisis: a phenomenon restricted to the interaction with the environment?

Authors:  Victor M Salinas-Torres; Rafael A Salinas-Torres; Ricardo M Cerda-Flores; Laura E Martínez-de-Villarreal
Journal:  Pediatr Surg Int       Date:  2018-03-17       Impact factor: 1.827

Review 3.  Familial occurrence of gastroschisis: a population-based overview on recurrence risk, sex-dependent influence, and geographical distribution.

Authors:  Victor M Salinas-Torres; Rafael A Salinas-Torres; Ricardo M Cerda-Flores; Laura E Martínez-de-Villarreal
Journal:  Pediatr Surg Int       Date:  2018-01-15       Impact factor: 1.827

4.  Effect of semaphorin 3C gene variants in multifactorial Hirschsprung disease.

Authors:  Fiko Ryantono; Raman Sethi; Alvin Santoso Kalim; Priscillia Imelda; Devy Melati; Susan Simanjaya; William Widitjiarso; Ririd Tri Pitaka; Nur Arfian; Kristy Iskandar; Akhmad Makhmudi; Poh San Lai
Journal:  J Int Med Res       Date:  2021-02       Impact factor: 1.671

  4 in total

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