Literature DB >> 26354986

Is normosmic Parkinson disease a unique clinical phenotype?

Dong H Lee1, Jungsu S Oh1, Jee H Ham1, Jae J Lee1, Injoo Lee1, Phil H Lee1, Jae S Kim2, Young H Sohn2.   

Abstract

OBJECTIVE: Olfactory dysfunction is present in the majority of patients with early-stage Parkinson disease (PD) and can precede the onset of motor symptoms by many years. We performed this study to evaluate whether normosmic patients with PD had different clinical features compared to hyposmic patients.
METHODS: We analyzed the data of 208 de novo patients with PD (mean age, 65.4 ± 9.7 years; range, 38-85 years; 104 men) who underwent both olfactory function tests and dopamine transporter (DAT) scans.
RESULTS: Normosmic patients were significantly younger and had fewer motor deficits than hyposmic patients with PD. Striatal subregional DAT activities were comparable between the 2 groups, but intersubregional gradients were significantly higher in normosmic than hyposmic PD. A general linear model showed that normosmic patients with PD showed significantly fewer motor deficits after controlling the patient's age, sex, symptom duration, and DAT activity in the posterior putamen (p = 0.016). Levodopa-equivalent dose at approximately 2.5 years follow-up tended to be lower in normosmic than in hyposmic PD (p = 0.055).
CONCLUSIONS: These results suggest that normosmic PD is a unique clinical phenotype with a more benign course, compared to hyposmic PD. Either less pathologic involvement in the olfactory system or a greater potential for olfactory neurogenesis in normosmic PD may contribute to this benign process compared to hyposmic PD.
© 2015 American Academy of Neurology.

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Year:  2015        PMID: 26354986     DOI: 10.1212/WNL.0000000000001999

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  19 in total

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Review 4.  60 Years of Achievements by KSNM in Neuroimaging Research.

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6.  Olfactory dysfunction and striatal dopamine transporter binding in motor subtypes of Parkinson's disease.

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Review 9.  Olfactory dysfunction in aging and neurodegenerative diseases.

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10.  Striatal Dopamine Depletion Patterns and Early Non-Motor Burden in Parkinsons Disease.

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