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Literature DB >> 26354901

The Complement Receptor C5aR Has a Dual, Time-Dependent Effect on the Outcome of Spinal Cord Injury.

Antigona Ulndreaj¹, Jessica M Marbourg², Pia M Vidal³.

Abstract

Entities: Disease Gene

Mesh：

Substances：

Year: 2015 PMID： 26354901 PMCID： PMC6605400 DOI： 10.1523/JNEUROSCI.2380-15.2015

Source DB: PubMed Journal: J Neurosci ISSN： 0270-6474 Impact factor: 6.167

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13 in total

1. Axonal plasticity and functional recovery after spinal cord injury in mice deficient in both glial fibrillary acidic protein and vimentin genes.

Authors: V Menet; M Prieto; A Privat; M Giménez y Ribotta
Journal: Proc Natl Acad Sci U S A Date: 2003-07-14 Impact factor: 11.205

2. Conditional ablation of Stat3 or Socs3 discloses a dual role for reactive astrocytes after spinal cord injury.

Authors: Seiji Okada; Masaya Nakamura; Hiroyuki Katoh; Tamaki Miyao; Takuya Shimazaki; Ken Ishii; Junichi Yamane; Akihiko Yoshimura; Yukihide Iwamoto; Yoshiaki Toyama; Hideyuki Okano
Journal: Nat Med Date: 2006-06-18 Impact factor: 53.440

3. Glial scar borders are formed by newly proliferated, elongated astrocytes that interact to corral inflammatory and fibrotic cells via STAT3-dependent mechanisms after spinal cord injury.

Authors: Ina B Wanner; Mark A Anderson; Bingbing Song; Jaclynn Levine; Ana Fernandez; Zachary Gray-Thompson; Yan Ao; Michael V Sofroniew
Journal: J Neurosci Date: 2013-07-31 Impact factor: 6.167

4. Delayed post-injury administration of C5a improves regeneration and functional recovery after spinal cord injury in mice.

Authors: Q Guo; J Cheng; J Zhang; B Su; C Bian; S Lin; C Zhong
Journal: Clin Exp Immunol Date: 2013-11 Impact factor: 4.330

5. Depletion of hematogenous macrophages promotes partial hindlimb recovery and neuroanatomical repair after experimental spinal cord injury.

Authors: P G Popovich; Z Guan; P Wei; I Huitinga; N van Rooijen; B T Stokes
Journal: Exp Neurol Date: 1999-08 Impact factor: 5.330

6. Quantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environment.

Authors: Kevin D Beck; Hal X Nguyen; Manuel D Galvan; Desirée L Salazar; Trent M Woodruff; Aileen J Anderson
Journal: Brain Date: 2010-01-19 Impact factor: 13.501

The Complement Receptor C5aR Has a Dual, Time-Dependent Effect on the Outcome of Spinal Cord Injury.

1. Axonal plasticity and functional recovery after spinal cord injury in mice deficient in both glial fibrillary acidic protein and vimentin genes.

2. Conditional ablation of Stat3 or Socs3 discloses a dual role for reactive astrocytes after spinal cord injury.

3. Glial scar borders are formed by newly proliferated, elongated astrocytes that interact to corral inflammatory and fibrotic cells via STAT3-dependent mechanisms after spinal cord injury.

4. Delayed post-injury administration of C5a improves regeneration and functional recovery after spinal cord injury in mice.

5. Depletion of hematogenous macrophages promotes partial hindlimb recovery and neuroanatomical repair after experimental spinal cord injury.

6. Quantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environment.

7. Complement C5 in experimental autoimmune encephalomyelitis (EAE) facilitates remyelination and prevents gliosis.

8. STAT3 is a critical regulator of astrogliosis and scar formation after spinal cord injury.

9. Reactive astrocytes protect tissue and preserve function after spinal cord injury.

10. The proinflammatory mediators C3a and C5a are essential for liver regeneration.