Literature DB >> 26349473

A phase I study of volasertib combined with afatinib, in advanced solid tumors.

Jean-Pascal Machiels1, Marc Peeters2, Catherine Herremans3, Veerle Surmont4, Pol Specenier2, Marina De Smet5, Korinna Pilz6,7, Natalja Strelkowa8, Dan Liu9, Sylvie Rottey10.   

Abstract

PURPOSE: To determine the maximum tolerated dose (MTD) of volasertib, a Polo-like kinase inhibitor, combined with afatinib, an oral irreversible ErbB family blocker, in patients with advanced solid tumors (NCT01206816; Study 1230.20).
METHODS: Patients with advanced non-resectable and/or metastatic disease following failure of conventional treatment received intravenous volasertib 150-300 mg on day 1 every 21 days, combined with oral afatinib 30-40 mg on days 2-21 of a 3-week cycle (Schedule A), or 50-90 mg on days 2-6 of a 3-week cycle (Schedule B). The primary objective was to determine the MTD of volasertib in combination with afatinib.
RESULTS: Fifty-seven patients (Schedule A, N = 29; Schedule B, N = 28) were treated. The MTDs were volasertib 300 mg plus afatinib 30 mg days 2-21 and 70 mg days 2-6 of a 3-week cycle for Schedules A and B, respectively. The most common Grade 3/4 adverse events were neutropenia (31.0 %), diarrhea (13.8 %), and thrombocytopenia (10.3 %) in Schedule A; neutropenia (39.3 %), thrombocytopenia (35.7 %), hypokalemia (14.3 %), febrile neutropenia, and nausea (each 10.7 %) in Schedule B. The best overall response was two partial responses (6.9 %; both in Schedule A); eight patients in each schedule achieved stable disease. Volasertib showed multi-exponential pharmacokinetic (PK) behavior; co-administration of volasertib and afatinib had no significant effects on the PK profile of either drug.
CONCLUSIONS: Volasertib combined with afatinib had manageable adverse effects and limited antitumor activity in this heavily pretreated population.

Entities:  

Keywords:  Advanced cancer; Afatinib; EGFR; PLK; Phase I trial; Volasertib

Mesh:

Substances:

Year:  2015        PMID: 26349473     DOI: 10.1007/s00280-015-2860-2

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  5 in total

1.  Volasertib suppresses the growth of human hepatocellular carcinoma in vitro and in vivo.

Authors:  Di-Wei Zheng; You-Qiu Xue; Yong Li; Jin-Ming Di; Jian-Ge Qiu; Wen-Ji Zhang; Qi-Wei Jiang; Yang Yang; Yao Chen; Meng-Ning Wei; Jia-Rong Huang; Kun Wang; Xing Wei; Zhi Shi
Journal:  Am J Cancer Res       Date:  2016-11-01       Impact factor: 6.166

2.  Volasertib suppresses tumor growth and potentiates the activity of cisplatin in cervical cancer.

Authors:  Feng-Feng Xie; Shi-Shi Pan; Rong-Ying Ou; Zhen-Zhen Zheng; Xiao-Xiu Huang; Meng-Ting Jian; Jian-Ge Qiu; Wen-Ji Zhang; Qi-Wei Jiang; Yang Yang; Wen-Feng Li; Zhi Shi; Xiao-Jian Yan
Journal:  Am J Cancer Res       Date:  2015-11-15       Impact factor: 6.166

Review 3.  PLK1 inhibition-based combination therapies for cancer management.

Authors:  Shengqin Su; Gagan Chhabra; Chandra K Singh; Mary A Ndiaye; Nihal Ahmad
Journal:  Transl Oncol       Date:  2021-12-29       Impact factor: 4.243

Review 4.  Present and Future Perspective on PLK1 Inhibition in Cancer Treatment.

Authors:  Michela Chiappa; Serena Petrella; Giovanna Damia; Massimo Broggini; Federica Guffanti; Francesca Ricci
Journal:  Front Oncol       Date:  2022-06-02       Impact factor: 5.738

5.  Polo-like kinase 1 (Plk1) inhibition synergizes with taxanes in triple negative breast cancer.

Authors:  Antonio Giordano; Yueying Liu; Kent Armeson; Yeonhee Park; Maya Ridinger; Mark Erlander; James Reuben; Carolyn Britten; Christiana Kappler; Elizabeth Yeh; Stephen Ethier
Journal:  PLoS One       Date:  2019-11-21       Impact factor: 3.240

  5 in total

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