Literature DB >> 26329470

Erratum: TMEM140 is associated with the prognosis of glioma by promoting cell viability and invasion.

Bin Li1, Ming-Zhu Huang2, Xiao-Qiang Wang3, Bang-Bao Tao1, Jun Zhong1, Xu-Hui Wang1, Wen-Chuan Zhang1, Shi-Ting Li4.   

Abstract

Entities:  

Year:  2015        PMID: 26329470      PMCID: PMC4556009          DOI: 10.1186/s13045-015-0199-0

Source DB:  PubMed          Journal:  J Hematol Oncol        ISSN: 1756-8722            Impact factor:   17.388


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It has been brought to our attention that in Fig. two of the article [1], the immunoblots in the middle panel were incorrectly labeled as TREM2 instead of TMEM140. The correct figure is included below (Fig. 1), in which the immunoblots in the middle panel are labeled as TMEM140. This labeling correction does not affect the results or interpretation of the results. We apologize for this error.
Fig. 1

Suppressing of TMEM140 expression by RNAi. a TMEM140 expression level in five glioma cell lines was analyzed by RT-PCR (left) and immunoblot (middle and right). b, c The effect of TMEM140 knockdown through siRNA silencing. The cells were transfected with normal control or TMEM140-RNAi for 48 h and then subjected to RT-PCR (left) and immunoblot analysis (middle and right) of the TMEM140 expression level. The representative images for immunoblot are shown in the middle panel, and data from three independent experiments were expressed as the mean ± S.D. (right panel). Wild type: wild-type cells; normal control: scrambled siRNA transfected cells; RNAi-1, RNAi-2, and RNAi-3: TMEM140-RNAi-1, -2, and -3 transfected cells (*P < 0.05, **P < 0.01, ***P < 0.001 compared with normal control)

Suppressing of TMEM140 expression by RNAi. a TMEM140 expression level in five glioma cell lines was analyzed by RT-PCR (left) and immunoblot (middle and right). b, c The effect of TMEM140 knockdown through siRNA silencing. The cells were transfected with normal control or TMEM140-RNAi for 48 h and then subjected to RT-PCR (left) and immunoblot analysis (middle and right) of the TMEM140 expression level. The representative images for immunoblot are shown in the middle panel, and data from three independent experiments were expressed as the mean ± S.D. (right panel). Wild type: wild-type cells; normal control: scrambled siRNA transfected cells; RNAi-1, RNAi-2, and RNAi-3: TMEM140-RNAi-1, -2, and -3 transfected cells (*P < 0.05, **P < 0.01, ***P < 0.001 compared with normal control)
  1 in total

1.  TMEM140 is associated with the prognosis of glioma by promoting cell viability and invasion.

Authors:  Bin Li; Ming-Zhu Huang; Xiao-Qiang Wang; Bang-Bao Tao; Jun Zhong; Xu-Hui Wang; Wen-Chuan Zhang; Shi-Ting Li
Journal:  J Hematol Oncol       Date:  2015-07-22       Impact factor: 17.388

  1 in total
  3 in total

Review 1.  Roles, molecular mechanisms, and signaling pathways of TMEMs in neurological diseases.

Authors:  Qinghong Chen; Junlin Fang; Hui Shen; Liping Chen; Mengying Shi; Xianbao Huang; Zhiwei Miao; Yating Gong
Journal:  Am J Transl Res       Date:  2021-12-15       Impact factor: 4.060

2.  Inhibition of Proliferation by Knockdown of Transmembrane (TMEM) 168 in Glioblastoma Cells via Suppression of Wnt/β-Catenin Pathway.

Authors:  Jie Xu; Zhongzhou Su; Qiuping Ding; Liang Shen; Xiaohu Nie; Xuyan Pan; Ai Yan; Renfu Yan; Yue Zhou; Liqin Li; Bin Lu
Journal:  Oncol Res       Date:  2019-03-25       Impact factor: 5.574

3.  TMC5 is Highly Expressed in Human Cancers and Corelates to Prognosis and Immune Cell Infiltration: A Comprehensive Bioinformatics Analysis.

Authors:  Hui Zhang; Xu Zhang; Weiguo Xu; Jian Wang
Journal:  Front Mol Biosci       Date:  2022-01-13
  3 in total

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