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Literature DB >> 26251225

Physiochemical artifacts in FeCl3 thrombosis models.

Keith B Neeves¹.
1. COLORADO SCHOOL OF MINES.

Abstract

In this issue of Blood, Ciciliano et al demonstrate that thrombus formation in ferric chloride (FeCl3) thrombosis models relies on physiochemical, rather than biological, mechanisms.

Entities: Chemical Disease

Mesh：

Substances：

Year: 2015 PMID： 26251225 DOI： 10.1182/blood-2015-05-644708

Source DB: PubMed Journal: Blood ISSN： 0006-4971 Impact factor: 22.113

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Cited

5 in total

1. Extrinsic sialylation is dynamically regulated by systemic triggers in vivo.

Authors: Charles T Manhardt; Patrick R Punch; Christopher W L Dougher; Joseph T Y Lau
Journal: J Biol Chem Date: 2017-07-17 Impact factor: 5.157

2. Elevated hematocrit enhances platelet accumulation following vascular injury.

Authors: Bethany L Walton; Marcus Lehmann; Tyler Skorczewski; Lori A Holle; Joan D Beckman; Jeremy A Cribb; Micah J Mooberry; Adam R Wufsus; Brian C Cooley; Jonathan W Homeister; Rafal Pawlinski; Michael R Falvo; Nigel S Key; Aaron L Fogelson; Keith B Neeves; Alisa S Wolberg
Journal: Blood Date: 2017-03-01 Impact factor: 22.113

Physiochemical artifacts in FeCl3 thrombosis models.

1. Extrinsic sialylation is dynamically regulated by systemic triggers in vivo.

2. Elevated hematocrit enhances platelet accumulation following vascular injury.

3. The Myosin II Inhibitor, Blebbistatin, Ameliorates FeCl3-induced Arterial Thrombosis via the GSK3β-NF-κB Pathway.

4. Ref-1 protects against FeCl₃-induced thrombosis and tissue factor expression via the GSK3β-NF-κB pathway.

5. Prevention of platelet aggregation and arterial thrombosis using a modified Shenzhu Guanxin Formula.

Physiochemical artifacts in FeCl3 thrombosis models.

1. Extrinsic sialylation is dynamically regulated by systemic triggers in vivo.

2. Elevated hematocrit enhances platelet accumulation following vascular injury.

3. The Myosin II Inhibitor, Blebbistatin, Ameliorates FeCl3-induced Arterial Thrombosis via the GSK3β-NF-κB Pathway.

4. Ref-1 protects against FeCl3-induced thrombosis and tissue factor expression via the GSK3β-NF-κB pathway.

5. Prevention of platelet aggregation and arterial thrombosis using a modified Shenzhu Guanxin Formula.

4. Ref-1 protects against FeCl₃-induced thrombosis and tissue factor expression via the GSK3β-NF-κB pathway.