Literature DB >> 26206100

Evaluation of an heterogeneous group of patients with von Willebrand disease using an assay alternative to ristocetin induced platelet agglutination.

F Stufano1, L Baronciani1, M T Pagliari1, F Franchi2, G Cozzi1, I Garcia-Oya1, P Bucciarelli1, M Boscarino1, F Peyvandi1,2.   

Abstract

BACKGROUND: Diagnosis of von Willebrand disease (VWD) type 2 usually relies on the discrepancy between the von Willebrand factor (VWF) ristocetin cofactor activity (VWF:RCo) and VWF antigen (VWF:Ag). Type 2B patients can be discriminated from other qualitative VWD variants by using ristocetin-induced platelet agglutination (RIPA) test. The major limitation of RIPA is the requirement of fresh blood sample.
OBJECTIVES: In this study, we evaluated the VWF gain-of-function mutant GPIb binding (VWF:GPIbM) and VWF:RCo assays to investigate whether the VWF:GPIbM/VWF:RCo ratio was able to identify the type 2B variant among an heterogeneous VWD population, previously characterized following the ISTH-SSC guidelines. PATIENTS/
METHODS: Seventy-six VWD patients and 31 healthy subjects were evaluated by using VWF:Ag, VWF:RCo, and VWF:GPIbM assays.
RESULTS: The mean (minimum-maximum values) VWF:GPIbM/VWF:RCo ratio was higher in type 2B patients (2.53, 0.84-6.11) than in healthy controls (1.05, 0.87-1.34), type 1 (0.85, 0.51-1.15), 2A (1.20, 0.36-2.82), and 2M (1.07, 0.91-1.38) (P < 0.0001). Type 2B variants were divided into four groups (A, B, C, and D) according to their different multimeric patterns. The mean value of the VWF:GPIbM/VWF:RCo ratio in the four groups showed an increasing trend from group A (1.08) to D (3.69), proportional to the loss of high molecular weight multimers. Among 32 type 2B patients, previously diagnosed with RIPA, 8 (mainly with a type I New York/Malmö phenotype) were not confirmed using the VWF:GPIbM/VWF:RCo ratio.
CONCLUSIONS: Whenever the RIPA test is not feasible, the VWF:GPIbM/VWF:RCo ratio might help to identify severe type 2B VWD patients.
© 2015 International Society on Thrombosis and Haemostasis.

Entities:  

Keywords:  GPIbalpha protein, human; ristocetin; von Willebrand disease, type 2; von Willebrand diseases; von Willebrand factor

Mesh:

Substances:

Year:  2015        PMID: 26206100     DOI: 10.1111/jth.13062

Source DB:  PubMed          Journal:  J Thromb Haemost        ISSN: 1538-7836            Impact factor:   5.824


  4 in total

Review 1.  Current challenges in the diagnosis and management of patients with inherited von Willebrand's disease in Italy: an Expert Meeting Report on the diagnosis and surgical and secondary long-term prophylaxis.

Authors:  Piercarla Schinco; Giancarlo Castaman; Antonio Coppola; Dorina Cultrera; Cosimo Ettorre; Anna C Giuffrida; Emanuela Marchesini; Renato Marino; Marta Milan; Claudio Molinari; Simona M Siboni; Ezio Zanon; Augusto B Federici
Journal:  Blood Transfus       Date:  2017-05-26       Impact factor: 3.443

2.  Rapid discrimination of the phenotypic variants of von Willebrand disease.

Authors:  Jonathan C Roberts; Patti A Morateck; Pamela A Christopherson; Ke Yan; Raymond G Hoffmann; Joan Cox Gill; Robert R Montgomery
Journal:  Blood       Date:  2016-02-25       Impact factor: 22.113

3.  Diagnosis and management of patients with von Willebrand's disease in Italy: an Expert Meeting Report.

Authors:  Flora Peyvandi
Journal:  Blood Transfus       Date:  2017-05-26       Impact factor: 3.443

4.  Comparison of von Willebrand factor platelet-binding activity assays: ELISA overreads type 2B with loss of HMW multimers.

Authors:  Attila Szederjesi; Luciano Baronciani; Ulrich Budde; Giancarlo Castaman; Paola Colpani; Andrew S Lawrie; Yuan Liu; Robert Montgomery; Flora Peyvandi; Reinhard Schneppenheim; Jürgen Patzke; Imre Bodó
Journal:  J Thromb Haemost       Date:  2020-08-27       Impact factor: 5.824

  4 in total

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