Literature DB >> 26201783

Oxidative stress is closely associated with insulin resistance in genotypes 1 and 3 chronic hepatitis C.

Said M Hashemi1, David van der Poorten1, Francisco Barrera1,2, Priyanka Bandara1, Ora Lux3, James Kench4, Jacob George5.   

Abstract

BACKGROUND: Chronic hepatitis C (CHC) infection is associated with insulin resistance and with oxidative stress, but the relationship between the two has not been thoroughly examined.
PURPOSE: To evaluate the association between insulin resistance and oxidative stress in CHC patients.
METHOD: In 115 CHC patients (68 with genotype 1 and 47 with genotype 3), the relationship between the serum concentration of malondialdehyde (MDA), a marker of oxidative stress and insulin resistance as defined by the homeostasis model (HOMA-IR) was examined.
RESULTS: There was no significant difference in MDA levels between genotype 1- and genotype 3-infected subjects (12.882 vs. 12.426 ng/mL, p = 0.2). By univariate analysis, factors associated with HOMA-IR in both genotypes were oxidative stress as measured by MDA (p = 0.002), body mass index (BMI), portal activity, and fibrosis. Genotype-specific differences in HOMA-IR association were steatosis and triglycerides (TG) for genotype 1, and age and glutathione (GSH) for genotype 3. In a stepwise multiple linear regression analysis in both genotypes, MDA was a significant and independent predictor of HOMA-IR (p = 0.04). As expected, BMI and fibrosis were likewise independently correlated to HOMA-IR. In addition, MDA levels were higher (p < 0.001) and GSH levels were lower (p = 0.023) in insulin-resistant subjects compared to their insulin-sensitive counterparts.
CONCLUSIONS: It is concluded that in CHC, oxidative stress is an independent predictor of HOMA-IR, irrespective of virus genotype. Further studies on the role of oxidative stress in the development of insulin resistance in CHC are warranted.

Entities:  

Keywords:  Hepatitis C; Insulin resistance; Malondialdehyde; Oxidative stress

Year:  2012        PMID: 26201783     DOI: 10.1007/s12072-012-9400-5

Source DB:  PubMed          Journal:  Hepatol Int        ISSN: 1936-0533            Impact factor:   6.047


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