Literature DB >> 26193985

The relationship between polymorphisms of genes regulating DNA repair or cell division and the toxicity of platinum and vinorelbine chemotherapy in advanced NSCLC patients.

T Powrózek1, R Mlak2, P Krawczyk3, I Homa3, M Ciesielka4, P Kozioł4, M Prendecka2, J Milanowski3, T Małecka-Massalska2.   

Abstract

INTRODUCTION: Platinum-based chemotherapy and 3rd generation drugs is still the main treatment option for advanced non-small cell lung cancer (NSCLC) patients without activating EGFR mutations or ALK rearrangements. However, the side effects associated with cytostatics are well known. Changes in the genes (e.g. single nucleotide polymorphisms, SNPs) encoding proteins regulating DNA repair or cell division could potentially influence on both the susceptibility of cancer cells to chemotherapy, and the occurrence of toxicities.
MATERIALS AND METHODS: In presented study, the relationship between the fourteen SNPs in nine DNA repair and cell division regulating genes: ERCC1, XPD, XPA, XPC, XRCC1, XPG, RRM1, BRCA1, STMN1 and the toxicity of first-line chemotherapy in NSCLC patients were investigated. SNPs were determined by SNaPshot PCR® in DNA isolated from peripheral blood of 55 NSCLC patients treated with platinum compound and vinorelbine. The toxicity of therapy was evaluated according to the Common Toxicity Criteria (CTC) Version 4.03.
RESULTS: The odds ratio (OR) of severe haematological toxicity was significantly lower in carriers of the T allele of XRCC1 gene (1196A > G, OR = 0.22, 95 % CI: 0.06-0.82, p = 0.018) and higher in the carriers of the T allele (2704C > A) of XPC gene (OR: 7.50, 95 % CI: 0.89-63.17, p = 0.036) compared to the remaining patients. Risk of severe hepatotoxicity was significantly lower in carriers of the C allele of STMN1 (-2166T > C, OR = 0.09, 95 % CI: 0.01-1.12, p = 0.025) than in patients with T allele of this gene. In carriers of G allele (2251A > C, OR: 0.24, 95 % CI: 0.07-0.81, p = 0.017) and T (934G > A, OR: 0.26, 95 % CI: 0.07-0.90, p = 0.029) of XPD gene, risk of severe nephrotoxicity was significantly lower than in other patients.
CONCLUSIONS: Selected SNPs of genes encoding DNA repair enzymes and cell division regulation proteins could be useful biomarkers for prediction of platinum and vinorelbine-based chemotherapy toxicity in patients with advanced NSCLC.

Entities:  

Keywords:  Chemotherapy; DNA repair; Non-small cell lung cancer; SNPs; STMN1; Toxicity

Mesh:

Substances:

Year:  2015        PMID: 26193985     DOI: 10.1007/s12094-015-1343-6

Source DB:  PubMed          Journal:  Clin Transl Oncol        ISSN: 1699-048X            Impact factor:   3.405


  25 in total

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Review 7.  Nucleotide excision repair: why is it not used to predict response to platinum-based chemotherapy?

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9.  Transcripts in pretreatment biopsies from a three-arm randomized trial in metastatic non-small-cell lung cancer.

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10.  Association of XPD polymorphisms with severe toxicity in non-small cell lung cancer patients in a Chinese population.

Authors:  Wenting Wu; Wei Zhang; Rong Qiao; Dan Chen; Huibo Wang; Yi Wang; Shuyu Zhang; Ge Gao; Aiqin Gu; Jie Shen; Ji Qian; Weiwei Fan; Li Jin; Baohui Han; Daru Lu
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Journal:  Pediatr Nephrol       Date:  2022-06-24       Impact factor: 3.714

2.  Association between DNA mismatch repair gene polymorphisms and platinum-based chemotherapy toxicity in non-small cell lung cancer patients.

Authors:  Jun-Yan Liu; Chen-Yue Qian; Yuan-Feng Gao; Juan Chen; Hong-Hao Zhou; Ji-Ye Yin
Journal:  Chin J Cancer       Date:  2017-01-16

3.  Genetic Variations and Cisplatin Nephrotoxicity: A Systematic Review.

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4.  A Pilot Study of Vinorelbine Safety and Pharmacokinetics in Patients with Varying Degrees of Liver Dysfunction.

Authors:  Jun Gong; May Cho; Rohan Gupta; Timothy W Synold; Paul Frankel; Christopher Ruel; Marwan Fakih; Vincent Chung; Dean Lim; Joseph Chao
Journal:  Oncologist       Date:  2019-02-01

5.  Genome-Wide Analyses of Nephrotoxicity in Platinum-Treated Cancer Patients Identify Association with Genetic Variant in RBMS3 and Acute Kidney Injury.

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