Literature DB >> 26186302

Iron Supplementation Associated With Loss of Phenotype in Autosomal Dominant Hypophosphatemic Rickets.

Klaus Kapelari1, Julia Köhle1, Dieter Kotzot1, Wolfgang Högler1.   

Abstract

CONTEXT: Autosomal dominant hypophosphatemic rickets (ADHR) is the only hereditary disorder of renal phosphate wasting in which patients may regain the ability to conserve phosphate. Low iron status plays a role in the pathophysiology of ADHR.
OBJECTIVE: This study reports of a girl with ADHR, iron deficiency, and a paternal history of hypophosphatemic rickets that resolved without treatment. The girl's biochemical phenotype resolved with iron supplementation.
SUBJECTS: A 26-month-old girl presented with typical features of hypophosphatemic rickets, short stature (79 cm; -2.82 SDS), and iron deficiency. Treatment with elemental phosphorus and calcitriol improved her biochemical profile and resolved the rickets. The girl's father had presented with rickets at age 11 months but never received medication. His final height was reduced (154.3 cm; -3.51 SDS), he had undergone corrective leg surgery and had an adult normal phosphate, fibroblast growth factor 23, and iron status. Father and daughter were found to have a heterozygous mutation in exon 3 of the FGF23 gene (c.536G>A, p.Arg179Gln), confirming ADHR. INTERVENTION: Withdrawal of rickets medication was attempted off and on iron supplementation.
RESULTS: Withdrawal of rickets medication in the girl was unsuccessful in the presence of low-normal serum iron levels at age 5.6 years but was later successful in the presence of high-normal serum iron levels following high-dose iron supplementation.
CONCLUSIONS: We report an association between iron supplementation and a complete loss of biochemical ADHR phenotype, allowing withdrawal of rickets medication. Experience from this case suggests that reduction and withdrawal of rickets medication should be attempted only after iron status has been optimized.

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Year:  2015        PMID: 26186302     DOI: 10.1210/jc.2015-2391

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  16 in total

Review 1.  Heritable and acquired disorders of phosphate metabolism: Etiologies involving FGF23 and current therapeutics.

Authors:  Erica L Clinkenbeard; Kenneth E White
Journal:  Bone       Date:  2017-01-31       Impact factor: 4.398

2.  A Rare Presentation of Rickets Mimicking Sacroiliitis: A Case Report and Literature Review.

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Journal:  Cureus       Date:  2022-06-17

3.  Systemic Control of Bone Homeostasis by FGF23 Signaling.

Authors:  Erica L Clinkenbeard; Kenneth E White
Journal:  Curr Mol Biol Rep       Date:  2016-02-03

4.  Oral Iron Replacement Normalizes Fibroblast Growth Factor 23 in Iron-Deficient Patients With Autosomal Dominant Hypophosphatemic Rickets.

Authors:  Erik A Imel; Ziyue Liu; Melissa Coffman; Dena Acton; Rakesh Mehta; Michael J Econs
Journal:  J Bone Miner Res       Date:  2019-10-25       Impact factor: 6.741

5.  Iron deficiency plays essential roles in the trigger, treatment, and prognosis of autosomal dominant hypophosphatemic rickets.

Authors:  C Liu; X Li; Z Zhao; Y Chi; L Cui; Q Zhang; F Ping; X Chai; Y Jiang; O Wang; M Li; X Xing; W Xia
Journal:  Osteoporos Int       Date:  2020-09-30       Impact factor: 4.507

Review 6.  FGF23 at the crossroads of phosphate, iron economy and erythropoiesis.

Authors:  Daniel Edmonston; Myles Wolf
Journal:  Nat Rev Nephrol       Date:  2019-09-13       Impact factor: 28.314

Review 7.  Congenital Conditions of Hypophosphatemia in Children.

Authors:  Erik Allen Imel
Journal:  Calcif Tissue Int       Date:  2020-04-23       Impact factor: 4.333

8.  Iron Chelation Resulting in Renal Phosphate Wasting.

Authors:  Lynda Cheddani; Thierry Leblanc; Caroline Silve; Nahid Tabibzadeh; Dominique Prié; Jean-Philippe Haymann; Marie-Noëlle Péraldi; Michel Daudon; Paul Meria; Emmanuel Letavernier
Journal:  Kidney Int Rep       Date:  2017-07-29

9.  The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1α expression.

Authors:  Julia M Hum; Erica L Clinkenbeard; Colin Ip; Taryn A Cass; Matt Allen; Kenneth E White
Journal:  Bone Rep       Date:  2017-01-17

10.  Under-recognized Hypoparathyroidism in Thalassemia

Authors:  Hataitip Tangngam; Pat Mahachoklertwattana; Preamrudee Poomthavorn; Ampaiwan Chuansumrit; Nongnuch Sirachainan; La-or Chailurkit; Patcharin Khlairit
Journal:  J Clin Res Pediatr Endocrinol       Date:  2018-05-04
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