Literature DB >> 26169058

Familial dysalbuminaemic hyperthyroxinaemia: a rapid and novel mass spectrometry approach to diagnosis.

Joshua B Ryan1, Stephen O Brennan1, Howard Potter1, Louise Wolmarans2, Christopher M Florkowski3, Peter M George1.   

Abstract

BACKGROUND: Familial dysalbuminaemic hyperthyroxinaemia is an important cause of discordant thyroid function test results (due to an inherited albumin variant); however, the diagnosis can be challenging. A 51-year-old man had persistently elevated free thyroxine (T4), with discordant normal thyroid-stimulating hormone and normal free triiodothyronine. He was clinically euthyroid and had a daughter with similar thyroid function test results. We aimed to apply a whole protein mass spectrometry method to investigate this case of suspected familial dysalbuminaemic hyperthyroxinaemia.
METHODS: Intact serum albumin was assessed directly using electrospray time-of-flight mass spectrometry. Results were confirmed using tryptic peptide m/z mapping and targeted DNA sequencing (exons 3 and 7 of the albumin gene). We also used this sequencing to screen 14 archived DNA samples that were negative for thyroid hormone receptor mutations (in suspected thyroid hormone resistance).
RESULTS: Mass spectrometry analysis demonstrated heterozygosity for an albumin variant with a 19 Da decrease in mass, indicative of an ArgHis substitution. The familial dysalbuminaemic hyperthyroxinaemia variant was confirmed with peptide mapping (showing the precise location of the substitution, 218Arg→His) and DNA sequencing (showing guanine to adenine transition at codon 218 of exon 7). The same familial dysalbuminaemic hyperthyroxinaemia variant was identified in one additional screened sample.
CONCLUSIONS: Time-of-flight mass spectrometry is a novel procedure for diagnosing familial dysalbuminaemic hyperthyroxinaemia. The test is rapid (<10 min), can be performed on <2 μL of serum and requires minimal sample preparation.
© The Author(s) 2015.

Entities:  

Keywords:  Familial dysalbuminaemic hyperthyroxinaemia; albumin variant; discordant thyroid function tests; mass spectrometry

Mesh:

Substances:

Year:  2015        PMID: 26169058     DOI: 10.1177/0004563215598168

Source DB:  PubMed          Journal:  Ann Clin Biochem        ISSN: 0004-5632            Impact factor:   2.057


  5 in total

1.  Serum Concentrations of Ubiquitin C-Terminal Hydrolase-L1 and Glial Fibrillary Acidic Protein after Pediatric Traumatic Brain Injury.

Authors:  Stefania Mondello; Firas Kobeissy; Annarita Vestri; Ronald L Hayes; Patrick M Kochanek; Rachel P Berger
Journal:  Sci Rep       Date:  2016-06-20       Impact factor: 4.379

Review 2.  Clinical, Genetic, and Protein Structural Aspects of Familial Dysalbuminemic Hyperthyroxinemia and Hypertriiodothyroninemia.

Authors:  Ulrich Kragh-Hansen; Monica Galliano; Lorenzo Minchiotti
Journal:  Front Endocrinol (Lausanne)       Date:  2017-11-01       Impact factor: 5.555

Review 3.  Finding effective biomarkers for pediatric traumatic brain injury.

Authors:  Olena Y Glushakova; Alexander V Glushakov; Ronald L Hayes
Journal:  Brain Circ       Date:  2016-10-18

4.  A Chinese Family with Familial Dysalbuminemic Hyperthyroxinemia (FDH) due to R242H Mutation on Human Albumin Gene: Reevaluating the Role of FDH in Patients with Asymptomatic Hyperthyroxinemia.

Authors:  Hongbing Liu; Jianmin Ran; Chuping Chen; Guangshu Chen; Ping Zhu; Rongshao Tan; Yan Liu
Journal:  Int J Endocrinol       Date:  2019-09-09       Impact factor: 3.257

5.  Familial Dysalbuminemic Hyperthyroxinemia as a Cause for Discordant Thyroid Function Tests.

Authors:  Matthew J M Ting; Rui Zhang; Ee Mun Lim; Bryan K Ward; Scott G Wilson; John P Walsh
Journal:  J Endocr Soc       Date:  2021-02-01
  5 in total

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