Literature DB >> 26133644

Leptin and metabolic syndrome in patients with Duchenne/Becker muscular dystrophy.

M Rodríguez-Cruz1, O R Cruz-Guzmán1, R E Escobar2, M López-Alarcón1.   

Abstract

OBJECTIVES: To determine whether patients with Duchenne/Becker muscular dystrophy (DMD/BMD) have components of metabolic syndrome (MetSy) and to evaluate whether leptin is associated with components of MetSy.
METHODS: This study included 78 patients (nine, <6 years of age; 54, 6 to <16 years of age; and 15 patients, ≥16 years of age). Obesity and body fat mass were determined by waist circumference and dual-energy X-ray absorptiometry, respectively. A 12-h fasting blood sample was collected in the morning. Patients were categorized into four groups according to the number of criteria for MetSy: group 0: none; group 1: one; group 2: two and group 3: three or more criteria.
RESULTS: All age groups showed components of MetSy. The concentration of these components was significantly higher in patients ≥16 years old. The prevalence of hypertriglyceridemia was from ~37% to 46% in all age groups. The prevalence of MetSy was 7.1% for patients from 6 to <16 years of age and 24% for patients ≥16 years of age. Serum leptin levels increased significantly (P < 0.05) with age; the highest (13.43 ± 9.4 ng/ml) value was observed in patients >16 years of age. Total leptin was correlated with the number of patients with MetSy (r = 0.383; P = 0.001).
CONCLUSIONS: Components of MetSy are significant in patients with DMD/BMD. A high prevalence of hypertriglyceridemia was observed. Younger patients with DMD/BMD have risk factors for MetSy. Although leptin increased according to different degrees of MetSy, this relation disappeared when the body fat was corrected by leptin; therefore, the association could be caused by a common risk factor-fat.
© 2015 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

Entities:  

Keywords:  duchenne; hypertriglyceridemia; leptin; metabolic syndrome; obesity

Mesh:

Substances:

Year:  2015        PMID: 26133644     DOI: 10.1111/ane.12450

Source DB:  PubMed          Journal:  Acta Neurol Scand        ISSN: 0001-6314            Impact factor:   3.209


  8 in total

1.  Longitudinal metabolomic analysis of plasma enables modeling disease progression in Duchenne muscular dystrophy mouse models.

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2.  Cross-sectional serum metabolomic study of multiple forms of muscular dystrophy.

Authors:  Pietro Spitali; Kristina Hettne; Roula Tsonaka; Ekrem Sabir; Alexandre Seyer; Jesse B A Hemerik; Jelle J Goeman; Esther Picillo; Manuela Ergoli; Luisa Politano; Annemieke Aartsma-Rus
Journal:  J Cell Mol Med       Date:  2018-02-14       Impact factor: 5.310

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Authors:  Pietro Spitali; Kristina Hettne; Roula Tsonaka; Mohammed Charrout; Janneke van den Bergen; Zaïda Koeks; Hermien E Kan; Melissa T Hooijmans; Andreas Roos; Volker Straub; Francesco Muntoni; Cristina Al-Khalili-Szigyarto; Marleen J A Koel-Simmelink; Charlotte E Teunissen; Hanns Lochmüller; Erik H Niks; Annemieke Aartsma-Rus
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6.  Hepatic Steatosis Assessment as a New Strategy for the Metabolic and Nutritional Management of Duchenne Muscular Dystrophy.

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7.  The Relationship between Obesity and Clinical Outcomes in Young People with Duchenne Muscular Dystrophy.

Authors:  Natassja Billich; Justine Adams; Kate Carroll; Helen Truby; Maureen Evans; Monique M Ryan; Zoe E Davidson
Journal:  Nutrients       Date:  2022-08-12       Impact factor: 6.706

8.  Plasma lipidomic analysis shows a disease progression signature in mdx mice.

Authors:  Roula Tsonaka; Alexandre Seyer; Annemieke Aartsma-Rus; Pietro Spitali
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  8 in total

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