Literature DB >> 26119093

Phase I study of combination of vorinostat, carboplatin, and gemcitabine in women with recurrent, platinum-sensitive epithelial ovarian, fallopian tube, or peritoneal cancer.

Ursula Matulonis1, Suzanne Berlin, Hang Lee, Christin Whalen, Elizabeth Obermayer, Richard Penson, Joyce Liu, Susana Campos, Carolyn Krasner, Neil Horowitz.   

Abstract

OBJECTIVES: Combining histone deacetylase inhibitors and chemotherapy is synergistic. This phase I study combined escalating vorinostat doses with constant doses of carboplatin and gemcitabine for the treatment of recurrent platinum-sensitive ovarian cancer. The objectives of this study were to determine the maximally tolerated dose of this combination; secondary objectives included preliminary response rate of this regimen and toxicity profile.
METHODS: Fifteen patients with relapsed ovarian cancer were enrolled into this phase I study. Doses of carboplatin and gemcitabine were AUC 4 on day 1 and 1000 mg/m(2) on days 1 and 8, respectively; cycles were administered every 21 days. Vorinostat was tested using four different schedules. The first dose level (DL A) tested vorinostat as daily oral dosing from days 1 to 14. DL B tested twice daily (BID) vorinostat dosing on days 1-3 and 8-10. DL C tested BID vorinostat dosing on days 1, 2, 8, and 9, starting vorinostat 1 day prior to initiation of carboplatin and gemcitabine, and DL D tested vorinostat on days 1 and 2 with chemotherapy starting on day 2.
RESULTS: All four DLs tested resulted in dose-limiting toxicities, and no MTD was determined. Toxicities were mostly hematologic. Seven patients were evaluable for RECIST assessment, and six of them had partial responses (PR) via RECIST.
CONCLUSIONS: Combination of carboplatin, gemcitabine, and vorinostat has activity in relapsed platinum-sensitive ovarian cancer, but was difficult to combine because of hematologic toxicities in this phase I study. No maximally tolerated dose was found, and the study was terminated early.

Entities:  

Mesh:

Substances:

Year:  2015        PMID: 26119093     DOI: 10.1007/s00280-015-2813-9

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  18 in total

1.  HDAC10 as a potential therapeutic target in ovarian cancer.

Authors:  Muhtadi M Islam; Tapahsama Banerjee; Colin Z Packard; Shweta Kotian; Karuppaiyah Selvendiran; David E Cohn; Jeffrey D Parvin
Journal:  Gynecol Oncol       Date:  2017-01-07       Impact factor: 5.482

Review 2.  Epigenetic Attire in Ovarian Cancer: The Emperor's New Clothes.

Authors:  Daniela Matei; Kenneth P Nephew
Journal:  Cancer Res       Date:  2020-05-07       Impact factor: 12.701

3.  Histone Deacetylase (HDAC) Inhibition Induces IκB Kinase (IKK)-dependent Interleukin-8/CXCL8 Expression in Ovarian Cancer Cells.

Authors:  Himavanth R Gatla; Yue Zou; Mohammad M Uddin; Bipradeb Singha; Pengli Bu; Ales Vancura; Ivana Vancurova
Journal:  J Biol Chem       Date:  2017-02-06       Impact factor: 5.157

Review 4.  DNA Methylation Biomarkers for Prediction of Response to Platinum-Based Chemotherapy: Where Do We Stand?

Authors:  Nuno Tiago Tavares; Saulė Gumauskaitė; João Lobo; Carmen Jerónimo; Rui Henrique
Journal:  Cancers (Basel)       Date:  2022-06-13       Impact factor: 6.575

5.  Development and Validation of an Inductively Coupled Plasma Mass Spectrometry (ICP-MS) Method for Quantitative Analysis of Platinum in Plasma, Urine, and Tissues.

Authors:  Ti Zhang; Shuang Cai; Wai Chee Forrest; Eva Mohr; Qiuhong Yang; M Laird Forrest
Journal:  Appl Spectrosc       Date:  2016-08-15       Impact factor: 2.388

6.  Beside P53 and PTEN: Identification of molecular alterations of the RAS/MAPK and PI3K/AKT signaling pathways in high-grade serous ovarian carcinomas to determine potential novel therapeutic targets.

Authors:  Shuhui Chen; Elisa Cavazza; Catherine Barlier; Julia Salleron; Pierre Filhine-Tresarrieu; Céline Gavoilles; Jean-Louis Merlin; Alexandre Harlé
Journal:  Oncol Lett       Date:  2016-09-02       Impact factor: 2.967

Review 7.  Epigenetic therapy for the treatment of epithelial ovarian cancer: A clinical review.

Authors:  Haller J Smith; J Michael Straughn; Donald J Buchsbaum; Rebecca C Arend
Journal:  Gynecol Oncol Rep       Date:  2017-03-21

8.  Influence of a novel histone deacetylase inhibitor panobinostat (LBH589) on the growth of ovarian cancer.

Authors:  Leslie A Garrett; Whitfield B Growdon; Bo R Rueda; Rosemary Foster
Journal:  J Ovarian Res       Date:  2016-09-15       Impact factor: 4.234

9.  Selective Inhibitors of Histone Deacetylases 1 and 2 Synergize with Azacitidine in Acute Myeloid Leukemia.

Authors:  Chengyin Min; Nathan Moore; Jeffrey R Shearstone; Steven N Quayle; Pengyu Huang; John H van Duzer; Matthew B Jarpe; Simon S Jones; Min Yang
Journal:  PLoS One       Date:  2017-01-06       Impact factor: 3.240

10.  Chromatin accessibility changes at intergenic regions are associated with ovarian cancer drug resistance.

Authors:  John Gallon; Erick Loomis; Edward Curry; Nicholas Martin; Leigh Brody; Ian Garner; Robert Brown; James M Flanagan
Journal:  Clin Epigenetics       Date:  2021-06-05       Impact factor: 6.551

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.