| Literature DB >> 26100488 |
Denise A Puga1, C Amy Tovar1, Zhen Guan1, John C Gensel2, Matthew S Lyman3, Dana M McTigue1, Phillip G Popovich1.
Abstract
All individuals experience stress and hormones (e.g., glucocorticoids/GCs) released during stressful events can affect the structure and function of neurons. These effects of stress are best characterized for brain neurons; however, the mechanisms controlling the expression and binding affinity of glucocorticoid receptors in the spinal cord are different than those in the brain. Accordingly, whether stress exerts unique effects on spinal cord neurons, especially in the context of pathology, is unknown. Using a controlled model of focal excitotoxic lower motor neuron injury in rats, we examined the effects of acute or chronic variable stress on spinal cord motor neuron survival and glial activation. New data indicate that stress exacerbates excitotoxic spinal cord motor neuron loss and associated activation of microglia. In contrast, hypertrophy and hyperplasia of astrocytes and NG2+ glia were unaffected or were modestly suppressed by stress. Although excitotoxic lesions cause significant motor neuron loss and stress exacerbates this pathology, overt functional impairment did not develop in the relevant forelimb up to one week post-lesion. These data indicate that stress is a disease-modifying factor capable of altering neuron and glial responses to pathological challenges in the spinal cord.Entities:
Keywords: Glucocorticoid receptor; Glucocorticoids; Glutamate; Hypothalamic–pituitary–adrenal axis; Microglia; Neuroinflammation; Spinal cord injury; Stress
Mesh:
Substances:
Year: 2015 PMID: 26100488 PMCID: PMC4567453 DOI: 10.1016/j.bbi.2015.06.006
Source DB: PubMed Journal: Brain Behav Immun ISSN: 0889-1591 Impact factor: 7.217