Literature DB >> 26064333

ATRA and Genistein synergistically inhibit the metastatic potential of human lung adenocarcinoma cells.

Ji Cheng1, Jun Qi2, Xue-Tao Li3, Kun Zhou1, Jing-Han Xu4, Yong Zhou1, Guo-Qiang Zhang5, Jian-Ping Xu6, Ren-Jie Zhou1.   

Abstract

OBJECTIVE: This study was to investigate the effects of all-trans retinoic acid (ATRA) in combination with Genistein on the proliferation, expression of apoptosis related proteins and adhesion molecules (MUC1 and ICAM-1) and invasiveness of A549 cells, aiming to investigate whether combined therapy of ATRA and Genistein is superior to monotherapy in suppressing metastasis of lung cancer cells.
METHODS: ATRA, Genistein and both were used to treat human lung adenocarcinoma cells (A549 cells). Immunohistochemistry was done for MUC1 expression, flow cytometry for ICAM-1 expression, fluorescence quantitative PCR for MUC1 expression and Western blot assay for the expressions of cell cycle related proteins (CDK4, Rb and p-ERK1/2) and apoptosis related proteins (Bax and Bcl-2). Cells were seeded into Matrigel pre-coated Transwell chambers, and the migrating cells were counted.
RESULTS: Combined treatment with ATRA and Genistein was able to reduce the expressions of Bcl-2, MUC1 and ICAM-1 and exerted synergistic effects to inhibit the invasion of A549 cells.
CONCLUSION: ATRA and Genistein may synergistically inhibit MUC1 and ICAM-1 expressions and affect the expressions of cell cycle related proteins (CDK4, Rb and p-ERK1/2) and apoptosis related proteins (Bax and Bcl-2), inhibit the metastatic potential of lung cancer A549 cells.

Entities:  

Keywords:  All-trans retinoic acid; Genistein; ICAM-1; MUC1; lung carcinoma; metastatic potential

Year:  2015        PMID: 26064333      PMCID: PMC4443167     

Source DB:  PubMed          Journal:  Int J Clin Exp Med        ISSN: 1940-5901


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