Edward H Yian1, Mark Dillon2, Jeff Sodl3, Emil Dionysian4, Ronald Navarro5, Anshuman Singh6. 1. Kaiser Permanente Southern California Orange County Kraemer Medical Offices, 3460 E. La Palma Avenue, Anaheim, CA 92806 USA. 2. Kaiser Permanente Northern California Sacramento Medical Center and Medical Offices, 2025 Morse Avenue, Sacramento, CA 95825 USA. 3. Kaiser Permanente Southern California Orange County Medical Office Building, 200 North Lewis Avenue, Orange, CA 92807 USA. 4. Kaiser Permanente Southern California Orange County Medical Office Building, 411 North Lakeview Avenue, Anaheim, CA 92807 USA. 5. Kaiser Permanente Southern California South Bay Medical Center, 25825 Vermont Avenue, Harbor City, CA 90710 USA. 6. Kaiser Permanente Southern California Orthopedics-Regular Clinic, Garfield Specialty Center, 5893 Copley Drive, San Diego, CA 92111 USA.
Abstract
BACKGROUND: The incidence of post-operative compressive peripheral neuropathy (CPN) after shoulder arthroplasty is not known. We hypothesized that the likelihood following shoulder arthroplasty would be higher compared to a non-operative cohort. METHODS: Retrospective study compared the incidence of symptomatic CPN after shoulder replacement to a 1:1 age- and gender-matched non-operative control group with shoulder arthritis. Six hundred six consecutive shoulder replacements from a regional shoulder arthroplasty registry were analyzed. This included 319 primary total shoulder arthroplasties (TSR), 168 hemiarthroplasties (HA), 31 humeral resurfacings (HHR), 71 reverse arthroplasties (RTSA), and 17 revision arthroplasties. Diagnosis of post-operative CPN was obtained by documented clinical examination by a physician consistent with CPN based on patient complaints, positive nerve study results, and/or nerve decompression. Age, gender, body mass index, diabetes status, thyroid abnormalities, operative side, and anesthesiology (ASA) score were examined. RESULTS: The surgery group had 15 cases (2.5 %) of post-operative CPN (ten carpal tunnel syndrome, five cubital tunnel syndrome). This included seven TSR, six HA, one revision TSR, and one RTSA. Diagnoses included ten osteoarthritis, four rotator cuff arthropathies, and one chondrolysis. Control group had eight cases (1.3 %) of CPN (seven carpal tunnel syndrome, one cubital tunnel syndrome). In univariate analysis, age, gender, body mass index, ASA score, operative side, thyroid status, and diabetes were not predictors of post-operative CPN. CPN incidence between surgical and control groups was not statistically significant. CONCLUSION: The 1-year incidence rate of new onset clinical post-operative CPN symptoms was 2.5 %. There was no significant difference of CPN rates between surgical and non-operative groups.
BACKGROUND: The incidence of post-operative compressive peripheral neuropathy (CPN) after shoulder arthroplasty is not known. We hypothesized that the likelihood following shoulder arthroplasty would be higher compared to a non-operative cohort. METHODS: Retrospective study compared the incidence of symptomatic CPN after shoulder replacement to a 1:1 age- and gender-matched non-operative control group with shoulder arthritis. Six hundred six consecutive shoulder replacements from a regional shoulder arthroplasty registry were analyzed. This included 319 primary total shoulder arthroplasties (TSR), 168 hemiarthroplasties (HA), 31 humeral resurfacings (HHR), 71 reverse arthroplasties (RTSA), and 17 revision arthroplasties. Diagnosis of post-operative CPN was obtained by documented clinical examination by a physician consistent with CPN based on patient complaints, positive nerve study results, and/or nerve decompression. Age, gender, body mass index, diabetes status, thyroid abnormalities, operative side, and anesthesiology (ASA) score were examined. RESULTS: The surgery group had 15 cases (2.5 %) of post-operative CPN (ten carpal tunnel syndrome, five cubital tunnel syndrome). This included seven TSR, six HA, one revision TSR, and one RTSA. Diagnoses included ten osteoarthritis, four rotator cuff arthropathies, and one chondrolysis. Control group had eight cases (1.3 %) of CPN (seven carpal tunnel syndrome, one cubital tunnel syndrome). In univariate analysis, age, gender, body mass index, ASA score, operative side, thyroid status, and diabetes were not predictors of post-operative CPN. CPN incidence between surgical and control groups was not statistically significant. CONCLUSION: The 1-year incidence rate of new onset clinical post-operative CPN symptoms was 2.5 %. There was no significant difference of CPN rates between surgical and non-operative groups.
Authors: M Mondelli; I Aprile; M Ballerini; F Ginanneschi; F Reale; C Romano; S Rossi; L Padua Journal: Eur J Neurol Date: 2005-12 Impact factor: 6.089
Authors: A Lädermann; A Lübbeke; B Mélis; R Stern; P Christofilopoulos; G Bacle; G Walch Journal: J Bone Joint Surg Am Date: 2011-07-20 Impact factor: 5.284