Literature DB >> 26019368

Snake bite and brain death - handle with care.

Subramanian Senthilkumaran1, Chidambaram Ananth2, Ritesh G Menezes3, Ponniah Thirumalaikolundusubramanian4.   

Abstract

Entities:  

Year:  2015        PMID: 26019368      PMCID: PMC4445165          DOI: 10.4103/0019-5049.156906

Source DB:  PubMed          Journal:  Indian J Anaesth        ISSN: 0019-5049


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Sir, The informative case report by Dayal et al.[1] has reinforced the need to search and look for bedside clinical clues to diagnose the underlying complications and/or status in snake bite. The patient cited[1] had both internal and external ophthalmoplegia, which mimicked brain death in many ways, and prompted the intensivist to withdraw ventilator support, with potential catastrophe. With regard to the case reported, we would like to add on to the clinical, diagnostic, therapeutic and behavioural aspects based on our earlier experiences.[2] In locked-in syndrome (LIS), the patient will exhibit features of quadriparesis and anarthria with preservation of consciousness, and retention of vertical eye movements, provided the patient is not in circulatory failure. The patients with LIS express non-verbal communication through vertical eye movements. In addition, frontalis muscle tends to escape even in severe cases of LIS.[3] The progression of the case[1] to the comatose state cannot be attributed simply to cerebral hypoxaemia, as the patient was well oxygenated. The deep and reversible coma is likely be due to the effects of the venom on the cortical and brain stem structures[4] and probably at synapses. Irreversible binding of venom to pre-synaptic portion makes clinical recovery slow in krait envenomation, as recovery occurs only with the formation of new neuromuscular junctions. In such cases, somatosensory evoked potential response with median nerve stimulation helps not only to diagnose the condition/status, but also to differentiate LIS from other neurological conditions. As the patients with LIS are invariably conscious and observe what is happening in and around, and hear the conversations, physicians and nurses handling such cases should be cautious of their words, deeds and communications so as to avoid conflicts between care providers and the beneficiary. In neurotoxic snake envenomation, paralysis by and large begins with proximal muscles first and then progresses to distal muscles invariably, whereas recovery occurs in the reverse order. Moreover, these cases require supportive measures till the effects of venom wear off, and a thorough assessment is made before embarking on next management options.
  4 in total

1.  Snake bite and brain death-cause for caution?

Authors:  Subramanian Senthilkumaran; Namasivayam Balamurugan; Ritesh G Menezes; Ponniah Thirumalaikolundusubramanian
Journal:  Am J Emerg Med       Date:  2013-02-04       Impact factor: 2.469

2.  Neurological aspects of ophitoxemia (Indian krait)- A clinico-electromyographic study.

Authors:  P K Sethi; J K Rastogi
Journal:  Indian J Med Res       Date:  1981-02       Impact factor: 2.375

3.  Deep coma and hypokalaemia of unknown aetiology following Bungarus caeruleus bites: Exploration of pathophysiological mechanisms with two case studies.

Authors:  Indika Bandara Gawarammana; Senanayake Abeysinghe Mudiyanselage Kularatne; Keerthi Kularatne; Roshita Waduge; Vajira Senaka Weerasinghe; Sunil Bowatta; Nimal Senanayake
Journal:  J Venom Res       Date:  2010-12-14

4.  Neurotoxin envenomation mimicking brain death in a child: A case report and review of literature.

Authors:  Madhu Dayal; Smita Prakash; Pradeep K Verma; Mridula Pawar
Journal:  Indian J Anaesth       Date:  2014-07
  4 in total
  1 in total

1.  Snakebite Mimicking Brain Death: Bedside Clues.

Authors:  Subramanian Senthilkumaran; Namasivayam Balamurugan; Nanjundan Karthikeyan; Ponniah Thirumalaikolundusubramanian
Journal:  Indian J Crit Care Med       Date:  2021-12
  1 in total

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