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Literature DB >> 26017778

Mad1 at the Golgi apparatus: a story beyond kinetochores.

Jun Wan¹, Beth A Weaver.

Abstract

Entities: Gene

Mesh：

Substances：

Year: 2015 PMID： 26017778 PMCID： PMC4613536 DOI： 10.1080/15384101.2015.1032639

Source DB: PubMed Journal: Cell Cycle ISSN： 1551-4005 Impact factor: 4.534

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7 in total

1. Genome-wide RNAi screening identifies human proteins with a regulatory function in the early secretory pathway.

Authors: Jeremy C Simpson; Brigitte Joggerst; Vibor Laketa; Fatima Verissimo; Cihan Cetin; Holger Erfle; Mariana G Bexiga; Vasanth R Singan; Jean-Karim Hériché; Beate Neumann; Alvaro Mateos; Jonathon Blake; Stephanie Bechtel; Vladimir Benes; Stefan Wiemann; Jan Ellenberg; Rainer Pepperkok
Journal: Nat Cell Biol Date: 2012-06-03 Impact factor: 28.824

2. Up-regulation of the mitotic checkpoint component Mad1 causes chromosomal instability and resistance to microtubule poisons.

Authors: Sean D Ryan; Eric M C Britigan; Lauren M Zasadil; Kristen Witte; Anjon Audhya; Avtar Roopra; Beth A Weaver
Journal: Proc Natl Acad Sci U S A Date: 2012-07-09 Impact factor: 11.205

1 in total

1. Mad1 destabilizes p53 by preventing PML from sequestering MDM2.

Authors: Jun Wan; Samuel Block; Christina M Scribano; Rebecca Thiry; Karla Esbona; Anjon Audhya; Beth A Weaver
Journal: Nat Commun Date: 2019-04-04 Impact factor: 14.919

1 in total

Mad1 at the Golgi apparatus: a story beyond kinetochores.

1. Genome-wide RNAi screening identifies human proteins with a regulatory function in the early secretory pathway.

2. Up-regulation of the mitotic checkpoint component Mad1 causes chromosomal instability and resistance to microtubule poisons.

3. The spindle checkpoint of budding yeast depends on a tight complex between the Mad1 and Mad2 proteins.

4. 2n or not 2n: Aneuploidy, polyploidy and chromosomal instability in primary and tumor cells.

5. A Golgi-localized pool of the mitotic checkpoint component Mad1 controls integrin secretion and cell migration.

6. Nuclear pores protect genome integrity by assembling a premitotic and Mad1-dependent anaphase inhibitor.

Review 7. Whole chromosome instability and cancer: a complex relationship.

1. Mad1 destabilizes p53 by preventing PML from sequestering MDM2.