Literature DB >> 25877044

The clinical features of autoimmunity in 53 patients with Wiskott-Aldrich syndrome in China: a single-center study.

Nan Chen1, Zhi-Yong Zhang2, Da-Wei Liu3, Wei Liu4, Xue-Mei Tang5, Xiao-Dong Zhao6.   

Abstract

UNLABELLED: Autoimmune disease (AD) is common in patients with Wiskott-Aldrich syndrome (WAS) and patients with WAS who has an AD usually constitute a high-risk group with poor outcome. However, knowledge of AD in WAS is limited in China. In this study, medical records of 53 patients with WAS at Children´s Hospital of Chongqing Medical University from April 2004 to January 2014 were evaluated retrospectively and 14 patients (26%) had at least one AD. Autoimmune hemolytic anemia (AIHA) was the most common and detected in 12 patients (23%), other complications included immune thrombocytopenia (n = 1), immune neutropenia (n = 1), autoimmune arthritis (n = 1), and renal injury (n = 1). No significant differences were found in the level of serum immunoglobulins and lymphocyte subsets between the AD group and non-AD group. Although eight patients with AD received hematopoietic stem cell transplantation (HSCT), three patients died of pulmonary infection after HSCT.
CONCLUSIONS: AD is frequent in Chinese patients with WAS and AIHA was the most common. AD is a poor prognosis factor for WAS and should be treated as early as possible by HSCT. WHAT IS KNOWN: • Autoimmune disease is common in patients with WAS. • Manifestations, follow-up finding, and treatment approaches of autoimmune disease in Chinese patients with WAS have received less attention in the literature. What is New: • This study is firstly intended for evaluation of the clinical and immune characteristics of autoimmune disease in a large series Chinese patients with WAS. • AD is frequent in Chinese patients with WAS and AIHA is the most common.

Entities:  

Keywords:  Autoimmune disease; Autoimmune hemolytic anemia; Hematopoietic stem cell transplantation; Wiskott–Aldrich syndrome

Mesh:

Substances:

Year:  2015        PMID: 25877044     DOI: 10.1007/s00431-015-2527-3

Source DB:  PubMed          Journal:  Eur J Pediatr        ISSN: 0340-6199            Impact factor:   3.183


  28 in total

1.  Development of IgA nephropathy-like glomerulonephritis associated with Wiskott-Aldrich syndrome protein deficiency.

Authors:  M Shimizu; N P Nikolov; K Ueno; K Ohta; R M Siegel; A Yachie; F Candotti
Journal:  Clin Immunol       Date:  2011-10-19       Impact factor: 3.969

2.  Renal transplantation in Wiskott-Aldrich syndrome.

Authors:  M C Webb; P A Andrews; C G Koffman; J S Cameron
Journal:  Transplantation       Date:  1993-12       Impact factor: 4.939

3.  The development of malignancy in the course of the Aldrich syndrome.

Authors:  R W ten Bensel; E M Stadlan; W Krivit
Journal:  J Pediatr       Date:  1966-05       Impact factor: 4.406

4.  Impaired interleukin-2 production in T-cells from a patient with Wiskott-Aldrich syndrome: basis of clinical effect of interleukin-2 replacement therapy.

Authors:  H Azuma; M Oshima; K Ito; A Okuno; I Kawabata; K Banba; H Murahashi; T Sekine; Y Kato; K Ikebuchi; H Ikeda
Journal:  Eur J Pediatr       Date:  2000-08       Impact factor: 3.183

Review 5.  Wiskott-Aldrich syndrome: a comprehensive review.

Authors:  Michel J Massaad; Narayanaswamy Ramesh; Raif S Geha
Journal:  Ann N Y Acad Sci       Date:  2013-03-25       Impact factor: 5.691

6.  Autoimmune diseases detected in children with primary immunodeficiency diseases: results from a reference centre at middle anatolia.

Authors:  Turkan Patiroglu; Hatice Eke Gungor; Ekrem Unal
Journal:  Acta Microbiol Immunol Hung       Date:  2012-09       Impact factor: 2.048

Review 7.  Stem cell transplantation for primary immune deficiency.

Authors:  Austen J J Worth; Claire Booth; Paul Veys
Journal:  Curr Opin Hematol       Date:  2013-11       Impact factor: 3.284

8.  Aberrant glycosylation of IgA in Wiskott-Aldrich syndrome and X-linked thrombocytopenia.

Authors:  Masaki Shimizu; Hirokazu Kanegane; Taizo Wada; Yaeko Motoyoshi; Tomohiro Morio; Fabio Candotti; Akihiro Yachie
Journal:  J Allergy Clin Immunol       Date:  2012-10-26       Impact factor: 10.793

9.  Characteristics and outcome of early-onset, severe forms of Wiskott-Aldrich syndrome.

Authors:  Nizar Mahlaoui; Isabelle Pellier; Cécile Mignot; Jean-Philippe Jais; Chrystèle Bilhou-Nabéra; Despina Moshous; Bénédicte Neven; Capucine Picard; Geneviève de Saint-Basile; Marina Cavazzana-Calvo; Stéphane Blanche; Alain Fischer
Journal:  Blood       Date:  2012-12-20       Impact factor: 22.113

10.  IgA nephropathy associated with X-linked thrombocytopenia.

Authors:  Hiro Matsukura; Hirokazu Kanegane; Kazushi Miya; Keisuke Ohtsubo; Akira Higuchi; Takakuni Tanizawa; Toshio Miyawaki
Journal:  Am J Kidney Dis       Date:  2004-03       Impact factor: 8.860

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  4 in total

Review 1.  Clinical Manifestations and Pathophysiological Mechanisms of the Wiskott-Aldrich Syndrome.

Authors:  Fabio Candotti
Journal:  J Clin Immunol       Date:  2017-10-30       Impact factor: 8.542

2.  Childhood-onset inflammatory bowel diseases associated with mutation of Wiskott-Aldrich syndrome protein gene.

Authors:  Takashi Ohya; Masakatsu Yanagimachi; Kentaro Iwasawa; Shuichiro Umetsu; Tsuyoshi Sogo; Ayano Inui; Tomoo Fujisawa; Shuichi Ito
Journal:  World J Gastroenterol       Date:  2017-12-28       Impact factor: 5.742

Review 3.  Clinical Features, Cancer Biology, Transplant Approach and Other Integrated Management Strategies for Wiskott-Aldrich Syndrome.

Authors:  Smitha Hosahalli Vasanna; Maria A Pereda; Jignesh Dalal
Journal:  J Multidiscip Healthc       Date:  2021-12-23

4.  A novel genomic inversion in Wiskott-Aldrich-associated autoinflammation.

Authors:  Immacolata Brigida; Samantha Scaramuzza; Dejan Lazarevic; Davide Cittaro; Francesca Ferrua; Lorena Leonardelli; Maria Alessio; Ornella Forma; Chiara Lanzani; Gianluca Viarengo; Fabio Ciceri; Momcilo Jankovic; Fernando Pesce; Alessandro Aiuti; Maria Pia Cicalese
Journal:  J Allergy Clin Immunol       Date:  2016-04-22       Impact factor: 10.793

  4 in total

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